Document Detail


Post-translational regulation of L-glutamic acid decarboxylase in the brain.
MedLine Citation:
PMID:  18270816     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Gamma-aminobutyric acid (GABA) is the primary inhibitory neurotransmitter in the central nervous system. GABA is converted from glutamic acid by the action of glutamic acid decarboxylase (GAD). There are two forms of GAD in the brain, GAD65 and GAD67, referring to a molecular weight of 65 and 67 kDa, respectively. Perturbations in GABAergic neurotransmission have been linked to a number of neurological disorders. Since GAD is the rate-limiting enzyme in controlling GABA synthesis, it is important to understand how GAD is regulated in the brain. It is known that GAD function can be regulated at the transcriptional/translational and post-translational levels. This review focuses briefly on the recent advances in revealing the post-translational regulation of GAD function including protein phosphorylation, palmitoylation and activity-dependent cleavage. The results from these studies have improved our understanding of the regulation of GAD function in the brain.
Authors:
Jianning Wei; Jang-Yen Wu
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review     Date:  2008-02-13
Journal Detail:
Title:  Neurochemical research     Volume:  33     ISSN:  1573-6903     ISO Abbreviation:  Neurochem. Res.     Publication Date:  2008 Aug 
Date Detail:
Created Date:  2008-06-30     Completed Date:  2008-11-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7613461     Medline TA:  Neurochem Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1459-65     Citation Subset:  IM    
Affiliation:
Department of Basic Science, Charles E. Schmidt College of Biomedical Science, Florida Atlantic University, Boca Raton, FL 33431, USA. jwei@fau.edu
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Sequence
Animals
Brain / enzymology*
Glutamate Decarboxylase / genetics,  metabolism*
Humans
Isoenzymes / genetics,  metabolism*
Molecular Sequence Data
Protein Processing, Post-Translational*
Sequence Alignment
gamma-Aminobutyric Acid / metabolism*
Grant Support
ID/Acronym/Agency:
NS37851/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Isoenzymes; 56-12-2/gamma-Aminobutyric Acid; EC 4.1.1.15/Glutamate Decarboxylase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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