Document Detail


Post-exercise lactate metabolism: a comparative review of sites, pathways, and regulation.
MedLine Citation:
PMID:  8815809     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Most vertebrates utilize supplemental lactate production to support the energetic demands of vigorous, brief exercise. Despite similar patterns of accumulation, there appears to be a trichotomy with regards to lactate processing post-exercise. Most fish retain most of their lactate intramuscularly, using it for in situ glycogen replenishment. Recent evaluation of fish muscle concludes that pyruvate kinase reversal is a probable gluconeogenic pathway. Amphibians and reptiles also utilize lactate as a muscle glyconeogenic substrate, but lactate is not sequestered post-exercise. None of these groups utilize hepatic gluconeogenesis to any significant extent post-exercise, and muscle glucose uptake is limited. Lactate oxidation plays a major role post-exercise in mammals, with hepatic and muscular gluco- and glyconeogenesis contributing to a lesser extent. Glucocorticoids may regulate lactate release from fish muscle, although catecholamines may influence glyconeogenesis in reptile muscle. Insulin affects lactate metabolism indirectly through its effects on muscle glucose metabolism.
Authors:
T T Gleeson
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Annual review of physiology     Volume:  58     ISSN:  0066-4278     ISO Abbreviation:  Annu. Rev. Physiol.     Publication Date:  1996  
Date Detail:
Created Date:  1996-10-04     Completed Date:  1996-10-04     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  0370600     Medline TA:  Annu Rev Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  565-81     Citation Subset:  IM    
Affiliation:
Environmental, Population, and Organismic Biology, University of Colorado, Boulder 80309-0334, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Exercise / physiology*
Humans
Lactates / metabolism*
Physical Exertion / physiology*
Grant Support
ID/Acronym/Agency:
R01 DK46408/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Lactates

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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