Document Detail


Positive-feedback loops in cell cycle progression.
MedLine Citation:
PMID:  19818353     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
A positive-feedback loop is a simple motif that is ubiquitous to the modules and networks that comprise cellular signaling systems. Signaling behaviors that are synonymous with positive feedback include amplification and rapid switching, maintenance, and the coherence of outputs. Recent advances have been made towards understanding how positive-feedback loops function, as well as their mechanistic basis in controlling eukaryotic cell cycle progression. Some of these advances will be reviewed here, including: how cyclin controls passage through Start and maintains coherence of G1/S regulon expression in yeast; how Polo-like kinase 1 activation is driven by Bora and Aurora A, and its expression is stimulated by Forkhead Box M1 in mammalian cells; and how some of the various dynamic behaviors of spindle assembly and anaphase onset can be produced.
Authors:
Joseph R Pomerening
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review     Date:  2009-10-07
Journal Detail:
Title:  FEBS letters     Volume:  583     ISSN:  1873-3468     ISO Abbreviation:  FEBS Lett.     Publication Date:  2009 Nov 
Date Detail:
Created Date:  2009-11-02     Completed Date:  2009-12-03     Revised Date:  2011-05-27    
Medline Journal Info:
Nlm Unique ID:  0155157     Medline TA:  FEBS Lett     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  3388-96     Citation Subset:  IM    
Affiliation:
Department of Biology, Interdisciplinary Program in Biochemistry, Indiana University, 212 S. Hawthorne Drive Simon Hall Room 043F, Bloomington, IN 47405-7003, United States. jpomeren@indiana.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Cycle* / genetics
Feedback, Physiological* / genetics
Gene Expression Regulation
Humans
Mitosis
Regulon / genetics
Grant Support
ID/Acronym/Agency:
R01 GM086526-01A1/GM/NIGMS NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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