| Portal vein phlebolithiasis found post-liver transplantation in the native liver of a child with biliary atresia. | |
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MedLine Citation:
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PMID: 11260490 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Biliary atresia is defined as partial or total obliteration of the extra-hepatic bile ducts. In advanced cases, liver transplantation (LTx) is considered the most appropriate treatment. This report describes a female patient whose biliary atresia and subsequent cirrhosis required LTx at 1 yr of age. Macroscopic inspection of the hilar region of the native liver post-Tx revealed the formation of a pouch in the hepatic duct and a stone in the lumen of the portal vein. X-ray diffraction analysis showed that the stone was composed of cholesteryl cinnamate, gluconic acid phenylhydrazide, Na beta broma-allyl mercaptomethyl penicillinate, and Al2O3 crystals. While the cholesterol component is a known element of gallstones, we attributed the Na beta broma-allyl mercaptomethyl penicillinate to the patient's drug therapy. Our literature search revealed no previous record or crystallographic analysis of portal vein phlebolithiasis. In this report we describe this rare finding. |
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Authors:
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B Bilezikçi; B Demirhan; H Kiyici; M Haberal |
Publication Detail:
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Type: Case Reports; Journal Article |
Journal Detail:
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Title: Pediatric transplantation Volume: 5 ISSN: 1397-3142 ISO Abbreviation: Pediatr Transplant Publication Date: 2001 Feb |
Date Detail:
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Created Date: 2001-03-30 Completed Date: 2001-05-10 Revised Date: 2004-11-17 |
Medline Journal Info:
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Nlm Unique ID: 9802574 Medline TA: Pediatr Transplant Country: Denmark |
Other Details:
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Languages: eng Pagination: 56-9 Citation Subset: IM |
Affiliation:
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Department of Pathology, Baskent University, Ankara, Turkey. banubie@yahoo.com |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Biliary Atresia
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complications*,
pathology,
surgery Calculi / chemistry, etiology* Female Humans Infant Jaundice / etiology, surgery Liver Transplantation* Portal Vein* Venous Thrombosis / complications* |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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