Document Detail


A portable hot spot recognition loop transfers sequence preferences from APOBEC family members to activation-induced cytidine deaminase.
MedLine Citation:
PMID:  19561087     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Enzymes of the AID/APOBEC family, characterized by the targeted deamination of cytosine to generate uracil within DNA, mediate numerous critical immune responses. One family member, activation-induced cytidine deaminase (AID), selectively introduces uracil into antibody variable and switch regions, promoting antibody diversity through somatic hypermutation or class switching. Other family members, including APOBEC3F and APOBEC3G, play an important role in retroviral defense by acting on viral reverse transcripts. These enzymes are distinguished from one another by targeting cytosine within different DNA sequence contexts; however, the reason for these differences is not known. Here, we report the identification of a recognition loop of 9-11 amino acids that contributes significantly to the distinct sequence motifs of individual family members. When this recognition loop is grafted from the donor APOBEC3F or 3G proteins into the acceptor scaffold of AID, the mutational signature of AID changes toward that of the donor proteins. These loop-graft mutants of AID provide useful tools for dissecting the biological impact of DNA sequence preferences upon generation of antibody diversity, and the results have implications for the evolution and specialization of the AID/APOBEC family.
Authors:
Rahul M Kohli; Shaun R Abrams; Kiran S Gajula; Robert W Maul; Patricia J Gearhart; James T Stivers
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural     Date:  2009-06-26
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  284     ISSN:  1083-351X     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2009 Aug 
Date Detail:
Created Date:  2009-08-17     Completed Date:  2009-10-06     Revised Date:  2010-09-24    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  22898-904     Citation Subset:  IM    
Affiliation:
Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205, USA.
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Motifs / genetics,  physiology
Amino Acid Sequence
Cytidine Deaminase / chemistry*,  genetics,  metabolism*
Cytosine Deaminase / chemistry,  genetics,  metabolism
Drug Resistance, Bacterial / genetics
Enzyme Inhibitors / pharmacology
Escherichia coli / drug effects,  genetics
Humans
Molecular Sequence Data
Mutation
Rifampin / pharmacology
Sequence Homology, Amino Acid
Grant Support
ID/Acronym/Agency:
GM056834-13/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 13292-46-1/Rifampin; EC 3.5.4.-/AICDA (activation-induced cytidine deaminase); EC 3.5.4.1/APOBEC3F protein, human; EC 3.5.4.1/Cytosine Deaminase; EC 3.5.4.5/APOBEC3G protein, human; EC 3.5.4.5/Cytidine Deaminase
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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