Document Detail


Polyunsaturated fatty acids are cerebral vasodilators via the TREK-1 potassium channel.
MedLine Citation:
PMID:  17556656     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Vessel occlusion is the most frequent cause for impairment of local blood flow within the brain resulting in neuronal damage and is a leading cause of disability and death worldwide. Polyunsaturated fatty acids and especially alpha-linolenic acid improve brain resistance against cerebral ischemia. The purpose of the present study was to evaluate the effects of polyunsaturated fatty acids and particularly alpha-linolenic acid on the cerebral blood flow and on the tone of vessels that regulate brain perfusion. alpha-Linolenic acid injections increased cerebral blood flow and induced vasodilation of the basilar artery but not of the carotid artery. The saturated fatty acid palmitic acid did not produce vasodilation. This suggested that the target of the polyunsaturated fatty acids effect was the TREK-1 potassium channel. We demonstrate the presence of this channel in basilar but not in carotid arteries. We show that vasodilations induced by the polyunsaturated fatty acid in the basilar artery as well as the laser-Doppler flow increase are abolished in TREK-1(-/-) mice. Altogether these data indicate that TREK-1 activation elicits a robust dilation that probably accounts for the increase of cerebral blood flow induced by polyunsaturated fatty acids such as alpha-linolenic acid or docosahexanoic acid. They suggest that the selective expression and activation of TREK-1 in brain collaterals could play a significant role in the protective mechanisms of polyunsaturated fatty acids against stroke by providing residual circulation during ischemia.
Authors:
Nicolas Blondeau; Olivier Pétrault; Stella Manta; Valérie Giordanengo; Pierre Gounon; Régis Bordet; Michel Lazdunski; Catherine Heurteaux
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-06-07
Journal Detail:
Title:  Circulation research     Volume:  101     ISSN:  1524-4571     ISO Abbreviation:  Circ. Res.     Publication Date:  2007 Jul 
Date Detail:
Created Date:  2007-07-20     Completed Date:  2007-11-13     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0047103     Medline TA:  Circ Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  176-84     Citation Subset:  IM    
Affiliation:
Institut de Pharmacologie Moléculaire et Cellulaire, UMR 6097, CNRS Université de Nice Sophia Antipolis, Institut Paul Hamel, Valbonne, France.
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MeSH Terms
Descriptor/Qualifier:
Animals
Blood Flow Velocity / drug effects
Brain Ischemia / metabolism*,  pathology,  physiopathology,  prevention & control*
Carotid Arteries / metabolism,  pathology,  physiopathology
Cerebellum / blood supply,  metabolism*,  pathology,  physiopathology
Cerebrovascular Circulation / drug effects
Gene Expression Regulation / drug effects*
Laser-Doppler Flowmetry
Mice
Mice, Knockout
Potassium Channels, Tandem Pore Domain / biosynthesis*,  deficiency
Rats
Regional Blood Flow / drug effects
Vasodilator Agents / pharmacology*
alpha-Linolenic Acid / pharmacology*
Chemical
Reg. No./Substance:
0/Potassium Channels, Tandem Pore Domain; 0/Vasodilator Agents; 0/potassium channel protein TREK-1; 463-40-1/alpha-Linolenic Acid
Comments/Corrections
Comment In:
Circ Res. 2007 Jul 20;101(2):119-21   [PMID:  17641234 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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