| Polymorphonuclear leucocyte function and previous yersinia arthritis: correlation of enhanced superoxide production with late manifestations. | |
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MedLine Citation:
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PMID: 2838004 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Polymorphonuclear leucocyte (PMN) functions (migration in vitro, chemiluminescence, O-2 production, and aggregation) were studied in 32 patients with previous yersinia arthritis (YA). PMNs of 11 HLA-B27 positive patients who had chronic or recurrent inflammatory symptoms showed O-2 production significantly higher than that of PMNs of 11 HLA-B27 positive patients without late manifestations. Also, PMNs of both HLA-B27 positive and negative patients tended to show chemotactic and chemokinetic migration rates higher than those of control cells of healthy HLA-B27 negative subjects. These functional aberrations may play a part in the development of the patients' inflammatory symptoms. |
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Authors:
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H Repo; P Koivuranta-Vaara; M Leirisalo-Repo |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't |
Journal Detail:
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Title: Annals of the rheumatic diseases Volume: 47 ISSN: 0003-4967 ISO Abbreviation: Ann. Rheum. Dis. Publication Date: 1988 Jun |
Date Detail:
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Created Date: 1988-07-21 Completed Date: 1988-07-21 Revised Date: 2009-11-18 |
Medline Journal Info:
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Nlm Unique ID: 0372355 Medline TA: Ann Rheum Dis Country: ENGLAND |
Other Details:
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Languages: eng Pagination: 452-7 Citation Subset: IM |
Affiliation:
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Department of Bacteriology and Immunology, University of Helsinki, Finland. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Adult Arthritis, Infectious / complications, immunology*, metabolism Chemotaxis, Leukocyte Female HLA Antigens / analysis HLA-B27 Antigen Humans Male Middle Aged Neutrophils / physiology* Superoxides / metabolism* Yersinia Infections / complications, immunology*, metabolism |
| Chemical | |
Reg. No./Substance:
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0/HLA Antigens; 0/HLA-B27 Antigen; 11062-77-4/Superoxides |
| Comments/Corrections | |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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