Document Detail


Polymorphisms predicted to alter function in prostaglandin E2 synthase and prostaglandin E2 receptors.
MedLine Citation:
PMID:  17460551     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND AND OBJECTIVE: Prostaglandin synthesis is the primary target of aspirin and other nonsteroidal antiinflammatory drugs, and thus is a pathway of major interest to pharmacology, pharmacogenetics, and epidemiology. Several lines of evidence implicate prostaglandin E2 in carcinogenesis; this study aimed to identify genetic variants in genes related to prostaglandin E2 synthesis and signaling. METHODS: We resequenced the coding regions of human prostaglandin E2 synthase (PGES), and prostaglandin E2 receptors EP1, EP2, and EP4 in 48 African-Americans and 47 Caucasians. RESULTS AND CONCLUSIONS: We identified 23 variants, 6 of which cause amino acid changes. The non-synonymous polymorphisms in PGES, EP1, and EP2 were present only among African-Americans; both populations carried non-synonymous polymorphisms in EP4. We used two sequence homology-based programs, SIFT and PolyPhen, to predict the impact of these polymorphisms. These programs predicted that the amino-acid changes p.Phe119Val in EP1, p.Ala44Glu in EP2, and possibly p.Val7Glu in PGES, p.Thr176Ile in EP4 and p.Gly420Asp in EP4 are likely to affect protein function. Thus, these variants may be relevant for inflammatory conditions, carcinogenesis, and pharmacogenetics.
Authors:
Jeannette Bigler; Justin G Sibert; Elizabeth M Poole; Christopher S Carlson; John D Potter; Cornelia M Ulrich
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  Pharmacogenetics and genomics     Volume:  17     ISSN:  1744-6872     ISO Abbreviation:  Pharmacogenet. Genomics     Publication Date:  2007 Mar 
Date Detail:
Created Date:  2007-04-26     Completed Date:  2007-05-18     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  101231005     Medline TA:  Pharmacogenet Genomics     Country:  United States    
Other Details:
Languages:  eng     Pagination:  221-7     Citation Subset:  IM    
Affiliation:
Cancer Prevention Program, Fred Hutchinson Cancer Research Center, Seattle, Washington 98109-1024, USA. jbigler@amgen.com
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MeSH Terms
Descriptor/Qualifier:
African Continental Ancestry Group / genetics
Amino Acid Substitution
Base Sequence
European Continental Ancestry Group / genetics
Forecasting
Gene Frequency
Humans
Intramolecular Oxidoreductases / genetics*,  physiology
Polymorphism, Genetic*
Receptors, Prostaglandin E / genetics*,  physiology
Sequence Analysis, Protein / methods
Sequence Homology, Amino Acid
Software
Structure-Activity Relationship
Grant Support
ID/Acronym/Agency:
R01 CA 89445/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Receptors, Prostaglandin E; EC 5.3.-/Intramolecular Oxidoreductases; EC 5.3.99.3/prostaglandin-E synthase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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