Document Detail


Polyglutamine neurodegeneration: protein misfolding revisited.
MedLine Citation:
PMID:  18778858     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Polyglutamine diseases are a major cause of neurodegeneration worldwide. Recent studies highlight the importance of protein quality control mechanisms in regulating polyglutamine-induced toxicity. Here we discuss a model of disease pathogenesis that integrates current understanding of the role of protein folding in polyglutamine disease with emerging evidence that alterations in native protein interactions contribute to toxicity. We also incorporate new findings on other age-related neurodegenerative diseases in an effort to explain how protein aggregation and normal aging processes might be involved in polyglutamine disease pathogenesis.
Authors:
Aislinn J Williams; Henry L Paulson
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review     Date:  2008-09-06
Journal Detail:
Title:  Trends in neurosciences     Volume:  31     ISSN:  0166-2236     ISO Abbreviation:  Trends Neurosci.     Publication Date:  2008 Oct 
Date Detail:
Created Date:  2008-09-23     Completed Date:  2009-04-09     Revised Date:  2011-06-01    
Medline Journal Info:
Nlm Unique ID:  7808616     Medline TA:  Trends Neurosci     Country:  England    
Other Details:
Languages:  eng     Pagination:  521-8     Citation Subset:  IM    
Affiliation:
Program in Neuroscience and Medical Scientist Training Program, University of Iowa, 2206 MERF, Iowa City, IA 52242, USA.
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MeSH Terms
Descriptor/Qualifier:
Aging / physiology
Animals
Humans
Models, Biological
Nerve Degeneration / genetics,  metabolism*
Neurotoxicity Syndromes / etiology,  pathology*
Peptides / metabolism*
Protein Folding*
Grant Support
ID/Acronym/Agency:
F31 NS056609-02/NS/NINDS NIH HHS; NS056609/NS/NINDS NIH HHS; NS38712/NS/NINDS NIH HHS; R01 NS038712-01A1/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Peptides; 26700-71-0/polyglutamine
Comments/Corrections

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