Document Detail


Platelet depletion in experimental myocardial infarction.
MedLine Citation:
PMID:  4026785     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Accumulation of platelets in the microvasculature after acute myocardial ischemia may exacerbate tissue injury through the formation of microthrombi and by the release of vasoactive substances. To assess the role of platelets in myocardial ischemic injury and infarction, circulating platelets were reduced by 94 +/- 2% (mean +/- S.E.M.) with sheep antiserum to canine platelets. Regional myocardial ischemia was produced by occlusion of the left circumflex coronary artery (LCCA) for 90 min followed by reperfusion for 5 hours. Infarct size did not differ significantly between antiplatelet serum and nonimmune serum groups: 36 +/- 8 vs. 43 +/- 4% of the area at risk, determined by a post-mortem dual staining technique (p greater than 0.05). A second occlusion-reperfusion control group, sacrificed at 24 hours, did not differ from 5 hr reperfused groups with regard to infarct size. Coronary sinus thromboxane B2 (TXB2) concentrations were not altered significantly by platelet depletion. Histopathologic examination confirmed the presence of necrosis in the infarcted myocardium and revealed substantial leukocytic infiltration in both groups. The results suggest that circulating platelets are not required for the full expression of myocardial ischemic injury resulting from temporary coronary artery occlusion followed by reperfusion.
Authors:
S R Jolly; W A Schumacher; S L Kunkel; G D Abrams; J Liddicoat; B R Lucchesi
Related Documents :
17221325 - Effects of low osmolar contrast (iomeprol) on haemorheology and platelet activation in ...
11994555 - Platelet glycoprotein ib alpha receptor polymorphisms and recurrent ischaemic events in...
3672435 - Unusual properties of platelet shape in coronary and cerebral artery disease.
19498175 - Dickkopf-1 enhances inflammatory interaction between platelets and endothelial cells an...
465125 - Platelet aggregation in relationship to plasma catecholamines in patients with hyperten...
10424375 - Mechanisms underlying increased platelet reactivity in patients with peripheral arteria...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Basic research in cardiology     Volume:  80     ISSN:  0300-8428     ISO Abbreviation:  Basic Res. Cardiol.     Publication Date:    1985 May-Jun
Date Detail:
Created Date:  1985-09-16     Completed Date:  1985-09-16     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0360342     Medline TA:  Basic Res Cardiol     Country:  GERMANY, WEST    
Other Details:
Languages:  eng     Pagination:  269-79     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Dogs
Hemodynamics
Male
Myocardial Infarction / blood*,  pathology
Myocardium / pathology
Platelet Aggregation*
Platelet Count*
Thromboxane A2 / metabolism
Thromboxane B2 / metabolism
Grant Support
ID/Acronym/Agency:
HL-19782/HL/NHLBI NIH HHS; HL-27817/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
54397-85-2/Thromboxane B2; 57576-52-0/Thromboxane A2

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Depletion of membrane phospholipid and mitochondrial dysfunction associated with coronary reperfusio...
Next Document:  Functional and reactive hyperemia are unaltered by homocysteine in conscious dogs.