Document Detail


Plasmacytoid dendritic cells in the skin: to sense or not to sense nucleic acids.
MedLine Citation:
PMID:  19250840     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Plasmacytoid dendritic cells (pDCs) are specialized sensors of viral nucleic acids that initiate protective immunity through the production of type I interferons (IFNs). Normally, pDCs fail to sense host-derived self-nucleic acids but do so when self-nucleic acids form complexes with endogenous antimicrobial peptides produced in damaged skin. Whereas regulated expression of antimicrobial peptides may lead to pDC activation and protective immune responses to skin injury, overexpression of antimicrobial peptides in psoriasis drives excessive sensing of self-nucleic acids by pDCs resulting in IFN-driven autoimmunity. In skin tumors, pDCs are unable to sense self-nucleic acids; however, therapeutic activation of pDCs by synthetic nucleic acids or analogues can be exploited to generate antitumor immunity.
Authors:
Curdin Conrad; Stephan Meller; Michel Gilliet
Publication Detail:
Type:  Journal Article; Review     Date:  2009-02-27
Journal Detail:
Title:  Seminars in immunology     Volume:  21     ISSN:  1096-3618     ISO Abbreviation:  Semin. Immunol.     Publication Date:  2009 Jun 
Date Detail:
Created Date:  2009-06-01     Completed Date:  2009-12-09     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9009458     Medline TA:  Semin Immunol     Country:  England    
Other Details:
Languages:  eng     Pagination:  101-9     Citation Subset:  IM    
Affiliation:
Department of Immunology, The University of Texas M.D. Anderson Cancer Center, 1515 Holcombe Blvd., Houston, TX 77030, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antigens, Differentiation / metabolism*
Autoimmunity
Cell Communication
Cell Differentiation
Cell Movement
Dendritic Cells / immunology*,  metabolism
Humans
Immunity, Innate*
Infection / immunology,  pathology
Interferon Type I / secretion*
Neoplasms / immunology,  pathology
Nucleic Acids / immunology*,  metabolism
Skin / immunology,  pathology
Toll-Like Receptors / metabolism
Chemical
Reg. No./Substance:
0/Antigens, Differentiation; 0/Interferon Type I; 0/Nucleic Acids; 0/Toll-Like Receptors

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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