Document Detail


Physiopathology of ventricular tachyarrhythmias.
MedLine Citation:
PMID:  6209647     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Ventricular arrhythmias may be the result of three mechanisms: abnormal automaticity, triggered activity complicating early or late after-depolarizations, and reentry by circular pathway or by reflection. These three fundamental mechanisms have been observed in the intact heart in experimental models of myocardial ischemia and digitalis intoxication. In man, the arguments in favor of a given mechanism are indirect and may be determined by their response to stimulation. It may be possible to state the following conclusions: (1) reentry is at the origin of ventricular fibrillation, certain ventricular tachycardias of bundle branch reentry and probably most chronic sustained ventricular tachycardias that are easily inducible; (2) the mechanism of certain other ventricular arrhythmias sustained remains unknown; (3) even when arrhythmias are associated with reentry, the triggering extrasystole can arise from a focal origin.
Authors:
G Motté; J F Laine; M Slama; C Sebag; J M Davy
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Pacing and clinical electrophysiology : PACE     Volume:  7     ISSN:  0147-8389     ISO Abbreviation:  Pacing Clin Electrophysiol     Publication Date:  1984 Nov 
Date Detail:
Created Date:  1985-01-24     Completed Date:  1985-01-24     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  7803944     Medline TA:  Pacing Clin Electrophysiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1129-36     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Action Potentials
Animals
Arrhythmias, Cardiac / physiopathology
Cardiac Pacing, Artificial
Chronic Disease
Coronary Disease / physiopathology
Disease Models, Animal
Dogs
Electrophysiology
Heart Ventricles / physiopathology
Humans
Myocardial Infarction / physiopathology
Swine
Tachycardia / physiopathology*

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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