Document Detail


Physiological reactivity to psychological stress in human pregnancy: current knowledge and future directions.
MedLine Citation:
PMID:  22800930     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cardiovascular and neuroendocrine reactivity to acute stress are important predictors of health outcomes in non-pregnant populations. Greater magnitude and duration of physiological responses have been associated with increased risk of hypertensive disorders and diabetes, greater susceptibility to infectious illnesses, suppression of cell-mediated immunity as well as risk for depression and anxiety disorders. Stress reactivity during pregnancy has unique implications for maternal health, birth outcomes, and fetal development. However, as compared to the larger literature, our understanding of the predictors and consequences of exaggerated stress reactivity in pregnancy is limited. This paper reviews the current state of this literature with an emphasis on gaps in knowledge and future directions.
Authors:
Lisa M Christian
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review     Date:  2012-07-16
Journal Detail:
Title:  Progress in neurobiology     Volume:  99     ISSN:  1873-5118     ISO Abbreviation:  Prog. Neurobiol.     Publication Date:  2012 Nov 
Date Detail:
Created Date:  2012-10-23     Completed Date:  2013-07-22     Revised Date:  2013-11-06    
Medline Journal Info:
Nlm Unique ID:  0370121     Medline TA:  Prog Neurobiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  106-16     Citation Subset:  IM    
Copyright Information:
Copyright © 2012 Elsevier Ltd. All rights reserved.
Affiliation:
Department of Psychiatry, The Ohio State University Medical Center, Columbus, OH 43210, USA. lisa.christian@osumc.edu
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MeSH Terms
Descriptor/Qualifier:
Female
Forecasting
Humans
Hypothalamo-Hypophyseal System / physiopathology
Neurosecretory Systems / physiopathology*
Predictive Value of Tests
Pregnancy / psychology*
Pregnancy Complications, Cardiovascular / etiology,  physiopathology*
Stress, Psychological / complications,  physiopathology*
Grant Support
ID/Acronym/Agency:
R21 HD061644/HD/NICHD NIH HHS; R21 HD067670/HD/NICHD NIH HHS; R21HD061644/HD/NICHD NIH HHS; R21HD067670/HD/NICHD NIH HHS; UL1 RR025755/RR/NCRR NIH HHS; UL1RR025755/RR/NCRR NIH HHS
Comments/Corrections

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