Document Detail


Phosphatidylserine exposure in Fas type I cells is mitochondria-dependent.
MedLine Citation:
PMID:  12804759     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Previous studies have demonstrated that Fas-triggered activation of effector caspases and subsequent nuclear apoptosis either is mitochondria-independent (type I cells) or relies on mitochondrial amplification of the initial stimulus (type II cells). We show herein that Bcl-2 overexpression in a prototypic type I cell line (SKW6.4) promotes mitochondrial generation of ATP and blocks Fas-triggered plasma membrane externalization of phosphatidylserine (PS). Moreover, overexpression of Bcl-2 attenuates macrophage engulfment of Fas-triggered cells. Fas-mediated DNA fragmentation, on the other hand, remains unaffected in SKW6.4-bcl-2 cells. These studies thus demonstrate that PS externalization and clearance of cell corpses are mitochondria-dependent events, and show that these events can be dissociated from other features of the apoptotic program, in Fas type I cells.
Authors:
Wanlaya Uthaisang; Leta K Nutt; Sten Orrenius; Bengt Fadeel
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  FEBS letters     Volume:  545     ISSN:  0014-5793     ISO Abbreviation:  FEBS Lett.     Publication Date:  2003 Jun 
Date Detail:
Created Date:  2003-06-13     Completed Date:  2003-07-25     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0155157     Medline TA:  FEBS Lett     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  110-4     Citation Subset:  IM    
Affiliation:
Institute of Environmental Medicine, Division of Toxicology, Nobels väg 13, Karolinska Institutet, 171 77, Stockholm, Sweden.
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MeSH Terms
Descriptor/Qualifier:
Adenosine Triphosphate / biosynthesis
Antibodies, Monoclonal / metabolism
Antigens, CD95 / metabolism*
Apoptosis
Caspases / metabolism
Cell Line
Cell Membrane / metabolism
DNA Fragmentation
Humans
Macrophages / metabolism
Membrane Proteins / metabolism
Mitochondria / metabolism*
Monocytes / cytology,  metabolism*
Phagocytosis
Phosphatidylserines / metabolism*
Proto-Oncogene Proteins c-bcl-2 / metabolism
Tumor Cells, Cultured
Chemical
Reg. No./Substance:
0/Antibodies, Monoclonal; 0/Antigens, CD95; 0/Membrane Proteins; 0/Phosphatidylserines; 0/Proto-Oncogene Proteins c-bcl-2; 56-65-5/Adenosine Triphosphate; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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