Document Detail


Phosphatidylinositol-specific-phospholipase C cleaves urokinase plasminogen activator receptor from the cell surface and leads to inhibition of pemphigus-IgG-induced acantholysis in DJM-1 cells, a squamous cell carcinoma line.
MedLine Citation:
PMID:  11422178     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We showed previously that pemphigus IgG enhanced both the activity of urokinase plasminogen activator (uPA) in cultured cells and the expression of its receptor (uPAR) on uPA-binding keratinocytes. In the present study, to clarify whether uPAR and uPA-activated plasmin are actually involved in the blistering process after pemphigus IgG binding to the cell surface, we examined the effects of the following on uPAR expression and on cell-cell detachment in DJM-1 cells, a squamous cell carcinoma line: (i) phosphatidylinositol-specific phospholipase C (PI-PLC) - which releases uPAR from the membrane surface into the culture medium by cleaving the glycosylphosphatidylinositol anchor thus inhibiting uPAR activity, and (ii) uPA inhibitors (tranexamic acid, aprotinin, p-aminobenzonic acid and dexamethasone). Preincubation with PI-PLC decreased dramatically the pemphigus IgG-induced uPAR expression in a dose-dependent manner, and inhibited pemphigus IgG-induced cell-cell detachment at 10 microg/mL. On the other hand, tranexamic acid (15 mM) inhibited pemphigus IgG-induced cell-cell detachment without reduction of uPAR expression, although aprotinin, p-aminobenzonic acid and dexamethasone failed to alter either of these parameters. Although uPAR expression on the pemphigus IgG-bound cell surface and uPA activation may contribute significantly to the pathogenesis of acantholysis in pemphigus, the mechanisms are complicated and should be defined further.
Authors:
S Asano; M Seishima; Y Kitajima
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Clinical and experimental dermatology     Volume:  26     ISSN:  0307-6938     ISO Abbreviation:  Clin. Exp. Dermatol.     Publication Date:  2001 May 
Date Detail:
Created Date:  2001-06-25     Completed Date:  2001-07-05     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  7606847     Medline TA:  Clin Exp Dermatol     Country:  England    
Other Details:
Languages:  eng     Pagination:  289-95     Citation Subset:  IM    
Affiliation:
Department of Dermatology, Gifu University School of Medicine, Gifu, Japan.
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MeSH Terms
Descriptor/Qualifier:
Acantholysis / immunology*
Humans
Immunoglobulin G / immunology*
Pemphigus / immunology*
Phosphatidylinositol Diacylglycerol-Lyase
Phosphoinositide Phospholipase C
Receptors, Cell Surface / drug effects,  metabolism*
Receptors, Urokinase Plasminogen Activator
Tumor Cells, Cultured
Type C Phospholipases / pharmacology*
Chemical
Reg. No./Substance:
0/Immunoglobulin G; 0/PLAUR protein, human; 0/Receptors, Cell Surface; 0/Receptors, Urokinase Plasminogen Activator; EC 3.1.4.-/Type C Phospholipases; EC 3.1.4.11/Phosphoinositide Phospholipase C; EC 4.6.1.13/Phosphatidylinositol Diacylglycerol-Lyase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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