Document Detail


Pharmacologic administration of interleukin-2.
MedLine Citation:
PMID:  20074271     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The development of biologic therapies for patients with cancer has in part been impeded by the extraordinary complexity and intrinsic feedback mechanisms promoting homeostasis in tissue injury, repair, inflammation, and immunity. Recombinant interleukin 2 (IL-2) therapy was initiated in 1984 based on its role as the prototypic T-cell growth factor, with novel roles deduced late after its FDA approval in regulating not only effector T cells but also regulatory T cells. Complicating its application, even in the most sophisticated centers, has been the manageable but difficult toxicities attendant on its use in spite of clear evidence of complete responses in 5-10% of treated patients with melanoma and renal cell carcinoma with extraordinary durability lasting now for almost 25 years, thus tantamount to "cures." Although efforts have been made to diminish toxicity or enhance efficacy the only substantive advance in combination therapy has been the application of tumor-infiltrating lymphocytes and the antibody to CTLA4. A deeper understanding of the "limiting" toxicity associated with mild flu-like symptoms and more debilitating cytokine "storm" not forthcoming. Here we propose the notion that the systemic syndrome associated with IL-2 administration is due to global cytokine-induced autophagy and temporally limited tissue dysfunction. The possible role of autophagy inhibitors to enhance efficacy and limit toxicity as well as possible problems with this approach are considered.
Authors:
Antonio Romo de Vivar Chavez; William Buchser; Per H Basse; Xiaoyan Liang; Leonard J Appleman; Jodi K Maranchie; Herbert Zeh; Michael E de Vera; Michael T Lotze
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Annals of the New York Academy of Sciences     Volume:  1182     ISSN:  1749-6632     ISO Abbreviation:  Ann. N. Y. Acad. Sci.     Publication Date:  2009 Dec 
Date Detail:
Created Date:  2010-01-15     Completed Date:  2010-01-22     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7506858     Medline TA:  Ann N Y Acad Sci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  14-27     Citation Subset:  IM    
Affiliation:
University of Pittsburgh Cancer Institute, Pennsylvania, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antineoplastic Agents / administration & dosage,  adverse effects,  therapeutic use
Autophagy / drug effects*
Humans
Interleukin-2 / administration & dosage,  adverse effects*,  metabolism,  therapeutic use
Neoplasms / diagnosis,  drug therapy
Prognosis
Grant Support
ID/Acronym/Agency:
1 P01 CA 101944-04/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Antineoplastic Agents; 0/Interleukin-2

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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