Document Detail


Pharmacokinetic self-potentiation of idarubicin by induction of anthracycline carbonyl reducing enzymes.
MedLine Citation:
PMID:  18398750     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Severe myelosuppression is one of the major adverse effects of Idarubicin (IDA). Especially, after two sequential therapy courses, IDA showed a stronger myelosuppression than after the first course. IDA was metabolized in liver by carbonyl reducing enzymes (CRE) to its 13-OH metabolite, idarubicinol(IDAol),which is more active compared with IDA. RLN-B2, a rat liver cell line, precultured in the presence of IDA showed higher CRE activity compared with non-precultured cells. A crude extract of the enzyme obtained from the livers of F344 rats preadministered IDA 7 days before they were sacrificed showed higher enzymatic activity than that from non-preadministered rats. At 4 h after IDA i.v., the production of IDAol was facilitated in the preadministered group compared with the non-preadministered group. In conclusion, CRE was induced by IDA pretreatment in vitro and vivo, resulting in increased IDAol, which could cause self-potentiation of the myelosuppressive and probably antitumor effects of IDA.
Authors:
Taro Yamashita; Toshihiro Fukushima; Takanori Ueda
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Leukemia & lymphoma     Volume:  49     ISSN:  1029-2403     ISO Abbreviation:  Leuk. Lymphoma     Publication Date:  2008 Apr 
Date Detail:
Created Date:  2008-04-09     Completed Date:  2008-08-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9007422     Medline TA:  Leuk Lymphoma     Country:  England    
Other Details:
Languages:  eng     Pagination:  809-14     Citation Subset:  IM    
Affiliation:
First Department of Internal Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Alcohol Oxidoreductases / genetics*,  metabolism
Animals
Anthracyclines
Cell Line
Gene Expression Regulation, Enzymologic / drug effects*
Idarubicin / metabolism,  pharmacokinetics*
Liver / cytology,  enzymology
Pharmacokinetics
Rats
Chemical
Reg. No./Substance:
0/Anthracyclines; 58957-92-9/Idarubicin; EC 1.1.-/Alcohol Oxidoreductases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Noxa mediates bortezomib induced apoptosis in both sensitive and intrinsically resistant mantle cell...
Next Document:  The DSM criteria of sexual dysfunction: need for a change.