Document Detail


Petrotetrayndiol A induces cell cycle arrest and apoptosis in SK-MEL-2 human melanoma cells through cytochrome c-mediated activation of caspases.
MedLine Citation:
PMID:  16458118     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We investigated the possible mechanisms by which petrotetrayndiol A, a polyacetylene from the sponge Petrosia sp., exerts its anti-proliferative activity in cultured SK-MEL-2 human melanoma cells. Petrotetrayndiol A-treated SK-MEL-2 cells showed growth inhibition and induction of apoptosis in a dose-dependent manner as measured by MTT assay, fluorescent microscopy and flow cytometric analysis. Flow cytometric analysis revealed that petrotetrayndiol A resulted in G2/M arrest in the cell cycle progression which was associated with a marked decrease in the protein expression of cyclin B1 and its activating partner Cdc2 with concomitant inductions of p21WAF1/CIP1. The increase in apoptosis was associated with a dose-dependent up-regulation of cytosolic factor, such as Bax and release of cytochrome c, and down-regulation of Bcl-2. We also observed activation of caspase-9 and caspase-3, DNA ladder formation, proteolytic degradation of poly(ADP-ribose)-polymerase (PARP), and selective down-regulation of cIAP-1. The apoptotic manifestations, such as PARP cleavage and DNA fragmentation, were abolished in the presence of the tripeptide caspase inhibitor z-VAD-fmk and a caspase-3-specific inhibitor Ac-DEVD-cho. Our data thus demonstrate that petrotetrayndiol A-induced apoptosis and growth inhibition of SK-MEL-2 cells is dependent on caspase activation.
Authors:
Hye Joung Choi; Su-Bog Yee; Sang Eun Park; Eunok Im; Jee Hyung Jung; Hae Young Chung; Yung Hyun Choi; Nam Deuk Kim
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2005-04-26
Journal Detail:
Title:  Cancer letters     Volume:  232     ISSN:  0304-3835     ISO Abbreviation:  Cancer Lett.     Publication Date:  2006 Feb 
Date Detail:
Created Date:  2006-02-06     Completed Date:  2006-04-06     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7600053     Medline TA:  Cancer Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  214-25     Citation Subset:  IM    
Affiliation:
Department of Pharmacy and Pusan Cancer Research Center, Pusan National University, Busan 609-735, South Korea.
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MeSH Terms
Descriptor/Qualifier:
Acetylene / analogs & derivatives*,  pharmacology
Animals
Antineoplastic Agents / pharmacology*
Apoptosis / drug effects*
Caspases / physiology*
Cell Cycle / drug effects*
Cell Line, Tumor
Cyclin-Dependent Kinase Inhibitor p21 / physiology
Cytochromes c / physiology*
Enzyme Activation
Humans
Inhibitor of Apoptosis Proteins / antagonists & inhibitors
Melanoma / drug therapy*,  pathology
Polyacetylenes
Porifera / chemistry*
Proto-Oncogene Proteins c-bcl-2 / analysis
Chemical
Reg. No./Substance:
0/Antineoplastic Agents; 0/CDKN1A protein, human; 0/Cyclin-Dependent Kinase Inhibitor p21; 0/Inhibitor of Apoptosis Proteins; 0/Proto-Oncogene Proteins c-bcl-2; 0/petrotetrayndiol A; 25067-58-7/Polyacetylenes; 74-86-2/Acetylene; 9007-43-6/Cytochromes c; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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