Document Detail


Perturbed bioelectrical properties of the mouse cecum following hepatectomy and starvation: the role of bacterial adherence.
MedLine Citation:
PMID:  10485604     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Previous work in our laboratory has demonstrated that bacterial adherence alone to the intestinal epithelium, as occurs following catabolic stress, significantly perturbs the normal electrophysiology of the cecal mucosa. The aim of this study was to further characterize these effects in the mouse cecum following hepatectomy and short-term starvation, and to define the role of bacterial adherence in this process. Groups of mice underwent a surgical hepatectomy and were either fed or starved during the postoperative period. Groups of controls underwent sham operations and were either fed or starved postoperatively. Electrophysiologic studies in Ussing chambers at 48 hours were performed. Bacterial adherence to the mucosa was assessed by culture and histologic staining. To determine the role of bacteria in the altered electrophysiologic response, ciprofloxacin decontamination studies were performed. Only mice subjected to both hepatectomy and starvation developed bacterial adherence of sufficient magnitude (>10(5) cfu/gm) to alter mucosal electrophysiology (short-circuit current and basal potential difference). Ciprofloxacin decontamination completely abrogated this effect. Ion replacement studies suggested that active sodium transport was primarily responsible for the observed changes in mucosal electrophysiology. Bacterial-epithelial cell interactions may be responsible for altered mucosal ion transport observed following operative catabolic stress and short-term starvation.
Authors:
J C Alverdy; B Hendrickson; S S Guandalini; R J Laughlin; K Kent; R Banerjee
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Shock (Augusta, Ga.)     Volume:  12     ISSN:  1073-2322     ISO Abbreviation:  Shock     Publication Date:  1999 Sep 
Date Detail:
Created Date:  1999-11-03     Completed Date:  1999-11-03     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9421564     Medline TA:  Shock     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  235-41; discussion 242     Citation Subset:  IM    
Affiliation:
Department of Surgery and Pediatrics, University of Chicago, IL 60637, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Bacterial Adhesion / physiology*
Cecum / immunology,  physiology*
Electrophysiology
Female
Hepatectomy
Immunoglobulin A / metabolism
Intestinal Mucosa / immunology,  physiology*
Ion Transport
Mice
Mice, Inbred BALB C
Starvation*
Grant Support
ID/Acronym/Agency:
DK26678/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Immunoglobulin A

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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