Document Detail


Peroxynitrite diminishes gap junctional communication: protection by selenite supplementation.
MedLine Citation:
PMID:  10632565     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Loss of intercellular communication via gap junctions has been correlated with progression of cells to a malignant phenotype. Here, we show that peroxynitrite, a mediator of toxicity in inflammatory processes, diminishes gap junctional intercellular communication (GJIC) in WB-F344 rat liver epithelial cells, assayed by the scrapeloading dye-transfer technique as well as by microinjection of a fluorescent dye into single cells. Exposure of cultured cells to a steady-state concentration of peroxynitrite of 1.6 microM for 4 min or to 3-morpholinosydnonimine (SIN-1) at 0.5 mM strongly diminished GJIC. These concentrations of peroxynitrite or SIN-1 were not cytotoxic. When cells were grown in a medium supplemented with sodium selenite (0.1-1 microM) for 72 h, substantial protection was afforded against the decrease in GJIC by peroxynitrite. Thus, peroxynitrite can disrupt GJIC, and selenium-containing proteins protect.
Authors:
V S Sharov; K Briviba; H Sies
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  IUBMB life     Volume:  48     ISSN:  1521-6543     ISO Abbreviation:  IUBMB Life     Publication Date:  1999 Oct 
Date Detail:
Created Date:  2000-02-09     Completed Date:  2000-02-09     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  100888706     Medline TA:  IUBMB Life     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  379-84     Citation Subset:  IM    
Affiliation:
Institut für Physiologische Chemie I, Heinrich-Heine-Universität Düsseldorf, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Communication / drug effects*
Cell Division / drug effects,  physiology
Cells, Cultured
Culture Media
Dose-Response Relationship, Drug
Epithelial Cells / cytology,  drug effects,  metabolism
Gap Junctions / drug effects*
Glutathione Peroxidase / biosynthesis
Homeostasis / drug effects
Liver / cytology,  drug effects,  metabolism
Nitrates / toxicity*
Oxidants / toxicity*
Protein Biosynthesis
Proteins*
Rats
Rats, Inbred F344
Selenoproteins
Sodium Selenite / metabolism,  pharmacology*
Chemical
Reg. No./Substance:
0/Culture Media; 0/Nitrates; 0/Oxidants; 0/Proteins; 0/Selenoproteins; 10102-18-8/Sodium Selenite; 26404-66-0/peroxynitric acid; EC 1.11.1.9/Glutathione Peroxidase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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