Document Detail


Peritoneal dialysate effluent during peritonitis induces human cardiomyocyte apoptosis by regulating the expression of GATA-4 and Bcl-2 families.
MedLine Citation:
PMID:  20625998     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cardiovascular event and infection are leading causes of death from peritoneal dialysis (PD). This study examined in vitro cellular mechanism for cardiotoxicity induced by PD-related peritonitis. Cultured human cardiomyocytes were treated with PD effluent (PDE) during peritonitis (PPDE), and effects of PPDE on cultured cardiomyocytes in terms of apoptosis, with expression its related genes assessed. Results showed PPDE treatment of cardiomyocyte leading to onset of apoptosis, as confirmed by phosphatidylserine exposure plus DNA fragmentation and damage. This apoptosis is mediated by reduced Bcl-2/Bax and Bcl-x(L)/Bax ratios, as well as reduced expression of GATA-4, an important cardiomyocyte survival factor, at the level of transcription. These changes activated pro-apoptotic pathways. PPDE treatment also inhibited ERK signals, contributing to cardiotoxicity. Our findings revealed that PPDE contains potent pro-apoptotic factors that regulate expression of GATA-4 and Bcl-2 families, inducing cultured cardiomyocyte apoptosis. This pinpoints a key role of apoptosis in PD-associated cardiovascular events, along with a potential therapeutic target.
Authors:
Hsin-Hui Wang; Ping-Chun Li; Hsiao-Ju Huang; Tzong-Yann Lee; Ching-Yuang Lin
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of cellular physiology     Volume:  226     ISSN:  1097-4652     ISO Abbreviation:  J. Cell. Physiol.     Publication Date:  2011 Jan 
Date Detail:
Created Date:  2010-10-27     Completed Date:  2010-12-06     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0050222     Medline TA:  J Cell Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  94-102     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Division of Nephrology, Taipei Veterans General Hospital, Taipei, Taiwan.
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*
Ascitic Fluid / physiology*
Cell Survival
Cells, Cultured
Dose-Response Relationship, Drug
Extracellular Signal-Regulated MAP Kinases / metabolism
Female
GATA4 Transcription Factor / genetics,  metabolism*
Gene Expression Regulation / drug effects
Genes, bcl-2 / genetics
Humans
Male
Middle Aged
Multigene Family
Myocytes, Cardiac / cytology,  drug effects*
Peritoneal Dialysis*
Peritonitis / pathology,  therapy
Proto-Oncogene Proteins c-bcl-2 / genetics,  metabolism*
Signal Transduction
bcl-2-Associated X Protein / genetics,  metabolism
Chemical
Reg. No./Substance:
0/GATA4 Transcription Factor; 0/GATA4 protein, human; 0/Proto-Oncogene Proteins c-bcl-2; 0/bcl-2-Associated X Protein; EC 2.7.11.24/Extracellular Signal-Regulated MAP Kinases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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