Document Detail


Peptide action in stroke therapy.
MedLine Citation:
PMID:  14519074     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Glutamate receptor antagonists, although effective in preventing in vitro excitotoxic death, also block the glutamatergic signalling that is essential for normal excitatory neurotransmission and neuronal survival. This has contributed to the failure of clinical trials employing glutamate receptor antagonists as stroke therapeutics. However, recent years have seen an increased understanding of the molecular organisation of glutamate receptors in the neuronal postsynaptic density. This and a dissection of their associated intracellular signalling cascades has allowed the identification of distinct pathways responsible for excitotoxicity. It has become possible to uncouple toxic signalling cascades from glutamate receptors by targeting the interactions of membrane receptors with downstream proteins. Toxic signalling can be effectively uncoupled from glutamate receptors using targeted, cell-permeable peptides to disrupt specific protein-protein interactions. This approach does not block essential excitatory neurotransmission, but attenuates neurotoxic signals specifically and reduces stroke damage. This novel approach to blocking excitotoxic signalling in cerebral ischaemia may constitute a practical approach to stroke therapy.
Authors:
Michelle M Aarts; Michael Tymianski
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Expert opinion on biological therapy     Volume:  3     ISSN:  1471-2598     ISO Abbreviation:  Expert Opin Biol Ther     Publication Date:  2003 Oct 
Date Detail:
Created Date:  2003-10-01     Completed Date:  2004-07-19     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  101125414     Medline TA:  Expert Opin Biol Ther     Country:  England    
Other Details:
Languages:  eng     Pagination:  1093-104     Citation Subset:  IM    
Affiliation:
Toronto Western Hospital, Suite 4W-435, 399 Bathurst Street, Toronto, Ontario, M5T 2S8, Canada.
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MeSH Terms
Descriptor/Qualifier:
Animals
Humans
Peptides / therapeutic use*
Receptors, Glutamate / drug effects
Signal Transduction / drug effects
Stroke / physiopathology,  therapy*
Chemical
Reg. No./Substance:
0/Peptides; 0/Receptors, Glutamate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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