Document Detail


Pathophysiology of ANCA-associated small vessel vasculitis.
MedLine Citation:
PMID:  20878509     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Antineutrophil cytoplasmic autoantibodies (ANCAs) directed to proteinase 3 (PR3-ANCA) or myeloperoxidase (MPO-ANCA) are strongly associated with the ANCA-associated vasculitides--Wegener's granulomatosis, microscopic polyangiitis, and Churg-Strauss syndrome. Clinical observations, including the efficacy of B-cell depletion via rituximab treatment, support--but do not prove--a pathogenic role for ANCA in the ANCA-associated vasculitides. In vitro experimental studies show that the interplay of ANCA, neutrophils, the alternative pathway of the complement system, and endothelial cells could result in lysis of the endothelium. A pathogenic role for MPO-ANCA is strongly supported by in vivo experimental studies in mice and rats, which also elucidate the pathogenic mechanisms involved in lesion development. Unfortunately, an animal model for PR3-ANCA-associated Wegener's granulomatosis is not yet available. Here, cellular immunity appears to play a major role as well, particularly via interleukin-17-producing T cells, in line with granulomatous inflammation in the lesions. Finally, microbial factors, in particular Staphylococcus aureus and gram-negative bacteria, seem to be involved in disease induction and expression, but further studies are needed to define their precise role in disease development.
Authors:
Cees G M Kallenberg
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Current rheumatology reports     Volume:  12     ISSN:  1534-6307     ISO Abbreviation:  Curr Rheumatol Rep     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-10-05     Completed Date:  2011-01-20     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100888970     Medline TA:  Curr Rheumatol Rep     Country:  United States    
Other Details:
Languages:  eng     Pagination:  399-405     Citation Subset:  IM    
Affiliation:
Department of Rheumatology and Clinical Immunology, University Medical Center Groningen, AA21, Hanzeplein 1, 9713 GZ, Groningen, The Netherlands. c.g.m.kallenberg@reuma.umcg.nl
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MeSH Terms
Descriptor/Qualifier:
Animals
Antibodies, Antineutrophil Cytoplasmic / blood*
Cells, Cultured
Disease Models, Animal
Humans
Immunity, Cellular / immunology
Mice
Microscopic Polyangiitis / immunology,  pathology,  physiopathology*
Myeloblastin / immunology
Neutrophils / cytology,  immunology
Peroxidase / immunology
Rats
Staphylococcus aureus / immunology,  pathogenicity
Th17 Cells / immunology
Chemical
Reg. No./Substance:
0/Antibodies, Antineutrophil Cytoplasmic; EC 1.11.1.7/Peroxidase; EC 3.4.21.76/Myeloblastin

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