Document Detail


Pathology of the thyroid in severe acute respiratory syndrome.
MedLine Citation:
PMID:  16996569     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The severe acute respiratory syndrome (SARS) epidemic started in November 2002 and spread worldwide. The pathological changes in several human organs of patients with SARS have been extensively described. However, to date, little has been reported about the effects of this infection on the thyroid gland. Femoral head necrosis and low serum triiodothyronine and thyroxine levels, commonly found in patients with SARS, raise the possibility of thyroid dysfunction. We have undertaken this study to evaluate for any potential injury to the thyroid gland caused by SARS on tissue samples obtained from 5 SARS autopsies. The terminal deoxynucleotidyl transferase-mediated dUPT nick end-labeling assay was performed to identify apoptotic cells. The follicular epithelium was found to be damaged with large numbers of cells exfoliated into the follicle. The terminal deoxynucleotidyl transferase-mediated dUPT nick end-labeling assay demonstrated many cells undergoing apoptosis. Follicular architecture was altered and showed distortion, dilatation, and collapse. No distinct calcitonin-positive cells were detectable in the SARS thyroids. In conclusion, both parafollicular and follicular cells were injured. This may provide an explanation both for low serum triiodothyronine and thyroxine levels and the osteonecrosis of the femoral head associated with patients with SARS. Apoptosis may play a role in the pathogenesis of SARS associated coronavirus infection in the thyroid gland.
Authors:
Lan Wei; Shen Sun; Cai-Hong Xu; Jing Zhang; Yun Xu; Hong Zhu; Suat-Cheng Peh; Christine Korteweg; Michael A McNutt; Jiang Gu
Related Documents :
9648109 - Measurement of cell death.
10965189 - Apoptosis in human term placenta. a morphological and gene expression study.
8840859 - Apoptosis of cardiac myocytes during cardiac allograft rejection. relation to induction...
15184579 - Oxidative and amphotericin b-mediated cell death in the opportunistic pathogen aspergil...
21160069 - Cytokine signature profiles in acquired aplastic anemia and myelodysplastic syndromes.
11063639 - Apoptosis may be an early event of progestin therapy for endometrial hyperplasia.
10489829 - Repetitive hyperpnoea causes peripheral airway obstruction and eosinophilia.
18424919 - Autophagy: an overlooked mechanism of hiv-1 pathogenesis and neuroaids?
17293979 - Cell proliferation and interferon-gamma response to recombinant mbp-3, narl, mt-10.3, a...
Publication Detail:
Type:  Case Reports; Journal Article; Research Support, Non-U.S. Gov't     Date:  2006-09-25
Journal Detail:
Title:  Human pathology     Volume:  38     ISSN:  0046-8177     ISO Abbreviation:  Hum. Pathol.     Publication Date:  2007 Jan 
Date Detail:
Created Date:  2006-12-15     Completed Date:  2007-02-15     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9421547     Medline TA:  Hum Pathol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  95-102     Citation Subset:  IM    
Affiliation:
Department of Anatomy, Histology and Embryology, School of Basic Medical Sciences, Peking (Beijing) University, 100083 Beijing, China.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adult
Apoptosis / physiology
Autopsy
Calcitonin / analysis
Fatal Outcome
Female
Humans
Immunohistochemistry
In Situ Nick-End Labeling
Male
Middle Aged
Severe Acute Respiratory Syndrome / complications*
Thyroid Diseases / etiology,  pathology*,  physiopathology
Thyroid Gland / chemistry,  pathology*,  physiopathology
Chemical
Reg. No./Substance:
9007-12-9/Calcitonin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Expression of alpha-methylacyl-coenzyme A racemase in dysplastic Barrett's epithelium.
Next Document:  The activated nerve growth factor receptor p-TrkA is selectively expressed in advanced-stage ovarian...