Document Detail


Pathology of hepatic iron overload.
MedLine Citation:
PMID:  17729397     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Although progress in imaging and genetics allow for a noninvasive diagnosis of most cases of genetic iron overload, liver pathology remains often useful (1) to assess prognosis by grading fibrosis and seeking for associated lesions and (2) to guide the etiological diagnosis, especially when no molecular marker is available. Then, the type of liver siderosis (parenchymal, mesenchymal or mixed) and its distribution throughout the lobule and the liver are useful means for suggesting its etiology: HLA-linked hemochromatosis gene (HFE) hemochromatosis or other rare genetic hemochromatosis, nonhemochromatotic genetic iron overload (ferroportin disease, aceruloplasminemia), or iron overload secondary to excessive iron supply, inflammatory syndrome, noncirrhotic chronic liver diseases including dysmetabolic iron overload syndrome, cirrhosis, and blood disorders.
Authors:
Yves Deugnier; Bruno Turlin
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  World journal of gastroenterology : WJG     Volume:  13     ISSN:  1007-9327     ISO Abbreviation:  World J. Gastroenterol.     Publication Date:  2007 Sep 
Date Detail:
Created Date:  2007-08-30     Completed Date:  2007-12-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100883448     Medline TA:  World J Gastroenterol     Country:  China    
Other Details:
Languages:  eng     Pagination:  4755-60     Citation Subset:  IM    
Affiliation:
Liver Unit and CIC INSERM 0203, Pontchaillou University Hospital, Rennes 35033, France. yves.deugnier@univ-rennes1.fr
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MeSH Terms
Descriptor/Qualifier:
Histocompatibility Antigens Class I / genetics
Humans
Iron Overload / diagnosis,  etiology*,  pathology*
Liver / metabolism,  pathology*
Liver Cirrhosis / metabolism,  pathology
Liver Diseases / complications
Membrane Proteins / genetics
Mutation / genetics
Chemical
Reg. No./Substance:
0/HFE protein, human; 0/Histocompatibility Antigens Class I; 0/Membrane Proteins

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