Document Detail


Pathologic effects of RNase-L dysregulation in immunity and proliferative control.
MedLine Citation:
PMID:  22202089     Owner:  NLM     Status:  In-Data-Review    
Abstract/OtherAbstract:
The endoribonuclease RNase-L is the terminal component of an RNA cleavage pathway that mediates antiviral, antiproliferative and immunomodulatory activities. Inactivation or dysregulation of RNase-L is associated with a compromised immune response and increased risk of cancer, accordingly its activity is tightly controlled and requires an allosteric activator, 2',5'-linked oligoadenylates, for enzymatic activity. The biological activities of RNase-L are a result of direct and indirect effects of RNA cleavage and microarray analyses have revealed that RNase-L impacts the gene expression program at multiple levels. The identification of RNase-L-regulated RNAs has provided insights into potential mechanisms by which it exerts antiproliferative, proapoptotic, senescence-inducing and innate immune activities. RNase-L protein interactors have been identified that serve regulatory functions and are implicated as alternate mechanisms of its biologic functions. Thus, while the molecular details are understood for only a subset of RNase-L activities, its regulation by small molecules and critical roles in host defense and as a candidate tumor suppressor make it a promising therapeutic target.
Authors:
Heather J Ezelle; Bret A Hassel
Related Documents :
18483419 - Modulation of the inflammatory response to streptococcus pneumoniae in a model of acute...
20952439 - The pathogenesis of idiopathic pulmonary fibrosis.
232689 - Alterations in lung macrophage antimicrobial activity associated with viral pneumonia.
12736389 - Meconium induces only localized inflammatory lung injury in piglets.
20298289 - Does tumour necrosis factor-alpha inhibitor infliximab induce histological resolution o...
20304959 - Ventilator-induced inflammatory response in lipopolysaccharide-exposed rat lung is medi...
20425759 - Systemic inflammatory load in humans is suppressed by consumption of two formulations o...
17222199 - Bortezomib disrupts tumour-dendritic cell interactions in myeloma and lymphoma: therape...
9292559 - Nitric oxide in renal health and disease.
Publication Detail:
Type:  Journal Article     Date:  2012-01-01
Journal Detail:
Title:  Frontiers in bioscience (Scholar edition)     Volume:  4     ISSN:  1945-0524     ISO Abbreviation:  Front Biosci (Schol Ed)     Publication Date:  2012  
Date Detail:
Created Date:  2011-12-28     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101485241     Medline TA:  Front Biosci (Schol Ed)     Country:  United States    
Other Details:
Languages:  eng     Pagination:  767-86     Citation Subset:  IM    
Affiliation:
Department of Microbiology and Immunology, University of Maryland School of Medicine, Baltimore MD.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  TGF-beta antiproliferative effects in tumor suppression.
Next Document:  The Role of tumor-associated macrophage in tumor progression.