Document Detail


Pathogenesis of gallstones.
MedLine Citation:
PMID:  20478481     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cholesterol gallstone formation is a complex process and involves phase separation of cholesterol crystals from supersaturated bile. In most cases, cholesterol hypersecretion is considered the primary event in gallstone formation. The sterol is transported through the hepatocytic canalicular membrane by ABCG5-G8. Expression of this transport protein is regulated by transcription factor Liver X Receptor-alpha, which may be responsible for biliary hypersecretion. Hydrophobic bile salt pool, bile concentration, excess pronucleating mucin, and impaired gallbladder and intestinal motility are secondary phenomena in most cases but nevertheless may contribute to gallstone formation.
Authors:
Niels Gerard Venneman; Karel Johannes van Erpecum
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Gastroenterology clinics of North America     Volume:  39     ISSN:  1558-1942     ISO Abbreviation:  Gastroenterol. Clin. North Am.     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-05-18     Completed Date:  2010-10-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8706257     Medline TA:  Gastroenterol Clin North Am     Country:  United States    
Other Details:
Languages:  eng     Pagination:  171-83, vii     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Inc. All rights reserved.
Affiliation:
Department of Gastroenterology and Hepatology, University Medical Center Utrecht, Utrecht, The Netherlands.
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MeSH Terms
Descriptor/Qualifier:
ATP-Binding Cassette Transporters / biosynthesis
Bile Acids and Salts / secretion*
Biliary Dyskinesia / complications*,  metabolism,  physiopathology
Biological Transport / physiology
Cholesterol / secretion*
Crystallization
Disease Progression
Gallbladder Emptying / physiology*
Gallstones* / etiology,  metabolism,  physiopathology
Humans
Lipoproteins / biosynthesis
Sterols / metabolism*
Chemical
Reg. No./Substance:
0/ABCG5 protein, human; 0/ABCG8 protein, human; 0/ATP-Binding Cassette Transporters; 0/Bile Acids and Salts; 0/Lipoproteins; 0/Sterols; 57-88-5/Cholesterol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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