Document Detail


Paraquat-induced Fanconi syndrome.
MedLine Citation:
PMID:  16221089     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The ingestion of paraquat, a non-selective herbicide, can be fatal in humans. Paraquat is toxic to multiple organs, including the kidney, heart, gastrointestinal tract and central nervous system. Although paraquat has been established as one cause of acute tubular necrosis, Fanconi syndrome presenting as severe hypophosphataemia after paraquat intoxication has not been reported. Here, we report the case of a 44-year-old Korean woman who presented with generalized proximal tubular dysfunction including aminoaciduria, phosphaturia and glycosuria after paraquat intoxication. We found that severe hypophosphataemia induces deep drowsiness. Renal biopsy findings indicated the presence of acute tubular necrosis that may be reversible.
Authors:
Hyo W Gil; Jong O Yang; Eun Y Lee; Sae Y Hong
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Publication Detail:
Type:  Case Reports; Journal Article    
Journal Detail:
Title:  Nephrology (Carlton, Vic.)     Volume:  10     ISSN:  1320-5358     ISO Abbreviation:  Nephrology (Carlton)     Publication Date:  2005 Oct 
Date Detail:
Created Date:  2005-10-13     Completed Date:  2005-12-15     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9615568     Medline TA:  Nephrology (Carlton)     Country:  Australia    
Other Details:
Languages:  eng     Pagination:  430-2     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine and Institute of Pesticide Poisoning, Soonchunhyang University Cheonan Hospital, Cheonan, Republic of Korea.
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MeSH Terms
Descriptor/Qualifier:
Adult
Biopsy
Fanconi Syndrome / chemically induced*,  pathology*
Female
Herbicides / poisoning*
Humans
Hypophosphatemia / chemically induced,  pathology
Kidney Tubular Necrosis, Acute / chemically induced,  pathology
Kidney Tubules, Proximal / drug effects,  pathology
Paraquat / poisoning*
Chemical
Reg. No./Substance:
0/Herbicides; 4685-14-7/Paraquat

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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