Document Detail

Pain threshold is achieved at intensity above anaerobic threshold in patients with intermittent claudication.
MedLine Citation:
PMID:  19770807     Owner:  NLM     Status:  MEDLINE    
PURPOSE: Walking training is considered as the first treatment option for patients with peripheral arterial disease and intermittent claudication (IC). Walking exercise has been prescribed for these patients by relative intensity of peak oxygen uptake (VO2peak), ranging from 40% to 70% VO2peak, or pain threshold (PT). However, the relationship between these methods and anaerobic threshold (AT), which is considered one of the best metabolic markers for establishing training intensity, has not been analyzed. Thus, the aim of this study was to compare, in IC patients, the physiological responses at exercise intensities usually prescribed for training (% VO2peak or % PT) with the ones observed at AT.
METHODS: Thirty-three IC patients performed maximal graded cardiopulmonary treadmill test to assess exercise tolerance. During the test, heart rate (HR), VO2, and systolic blood pressure were measured and responses were analyzed at the following: 40% of VO2peak; 70% of VO2peak; AT; and PT.
RESULTS: Heart rate and VO2 at 40% and 70% of VO2peak were lower than those at AT (HR: -13 +/- 9% and -3 +/- 8%, P < .01, respectively; VO2: -52 +/- 12% and -13 +/- 15%, P < .01, respectively). Conversely, HR and VO2 at PT were slightly higher than those at AT (HR: +3 +/- 8%, P < .01; VO2: +6 +/- 15%, P = .04). None of the patients achieved the respiratory compensation point.
CONCLUSION: Prescribing exercise for IC patients between 40% and 70% of VO2peak will induce a lower stimulus than that at AT, whereas prescribing exercise at PT will result in a stimulus above AT. Thus, prescribing exercise training for IC patients on the basis of PT will probably produce a greater metabolic stimulus, promoting better cardiovascular benefits.
Raphael Mendes Ritti-Dias; Cláudia Lúcia de Moraes Forjaz; Gabriel Grizzo Cucato; Luis Augusto Riani Costa; Nelson Wolosker; Maria de Fátima Nunes Marucci
Related Documents :
19699847 - Mechanisms of exercise intolerance in patients with hypertrophic cardiomyopathy.
19568197 - Dietary arginine supplementation speeds pulmonary vo2 kinetics during cycle exercise.
8455457 - Oxygen consumption using the k2 telemetry system and a metabolic cart.
11179407 - The slow component of o(2) uptake is not accompanied by changes in muscle emg during re...
8336357 - Participation in community sports centres: motives and predictors of enjoyment.
15110377 - Anesthesiology: perioperative medicine or "when the anesthetic is a diuretic".
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of cardiopulmonary rehabilitation and prevention     Volume:  29     ISSN:  1932-751X     ISO Abbreviation:  J Cardiopulm Rehabil Prev     Publication Date:    2009 Nov-Dec
Date Detail:
Created Date:  2009-11-26     Completed Date:  2011-01-25     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101291247     Medline TA:  J Cardiopulm Rehabil Prev     Country:  United States    
Other Details:
Languages:  eng     Pagination:  396-401     Citation Subset:  IM    
Department of Nutrition, School of Public Health, Exercise Hemodynamic Laboratory, School of Physical Education and Sport, University of São Paulo, São Paulo, Brazil.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Anaerobic Threshold*
Analysis of Variance
Ankle Brachial Index
Cross-Sectional Studies
Exercise Test
Exercise Therapy
Exercise Tolerance
Heart Rate
Intermittent Claudication / physiopathology*,  therapy
Motor Activity
Oxygen Consumption
Peripheral Arterial Disease / physiopathology*,  therapy

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

Previous Document:  Physical exercise improves the peripheral microcirculation of patients with chronic heart failure.
Next Document:  Patient and program outcome assessment in pulmonary rehabilitation: an AACVPR statement.