Document Detail


PML-RAR{alpha} and Dnmt3a1 cooperate in vivo to promote acute promyelocytic leukemia.
MedLine Citation:
PMID:  20861188     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The PML-RARα oncogene is the central effector of acute promyelocytic leukemia (APL). PML-RARα physically interacts with epigenetic-modifying enzymes including DNA methyltransferases (Dnmt) to suppress critical downstream targets. Here, we show that increased expression of Dnmt3a1 cooperates with PML-RARα in vivo to promote early lethality secondary to myeloid expansion and dysfunction in primary mice. Bone marrow cells from these mice cause leukemogenesis with a shortened latency and a higher penetrance on transplantation into irradiated recipients. Furthermore, leukemic cells overexpressing PML-RARα and Dnmt3a1 display increased methylation at a target promoter compared with PML-RARα or Dnmt3a1 controls. Our findings show a cooperation between the PML-RARα oncogene and the Dnmt3a1 enzyme in vivo and that Dnmt levels can be rate limiting in APL progression.
Authors:
Deepa Subramanyam; Cassandra D Belair; Keegan Q Barry-Holson; Haijiang Lin; Scott C Kogan; Emmanuelle Passegué; Robert Blelloch
Related Documents :
12808098 - Cooperation of cytokine signaling with chimeric transcription factors in leukemogenesis...
12022398 - Outbreak of hind limb paralysis in young cfw swiss webster mice.
20647568 - A protease-resistant pml-rar{alpha} has increased leukemogenic potential in a murine mo...
23585228 - Impairment of autophagy decreases ventilator-induced lung injury by blockade of the nfÎ...
9003248 - Analysis of b7-1 and b7-2 costimulatory ligands in cultured mouse microglia: upregulati...
17089128 - Blockade of stat3 by antisense oligonucleotide in tnbs-induced murine colitis.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-09-21
Journal Detail:
Title:  Cancer research     Volume:  70     ISSN:  1538-7445     ISO Abbreviation:  Cancer Res.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-11-02     Completed Date:  2010-12-21     Revised Date:  2013-05-27    
Medline Journal Info:
Nlm Unique ID:  2984705R     Medline TA:  Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  8792-801     Citation Subset:  IM    
Copyright Information:
©2010 AACR.
Affiliation:
Institute for Regeneration Medicine, University of California, San Francisco, California 94143, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Bone Marrow Cells / pathology
DNA (Cytosine-5-)-Methyltransferase / genetics*
DNA Methylation*
Flow Cytometry
Genes, Lethal*
Humans
Inflammation / etiology,  pathology
Leukemia, Promyelocytic, Acute / etiology*,  pathology
Mice
Mice, Transgenic
Oncogene Proteins, Fusion / genetics*
Promoter Regions, Genetic / genetics
Respiratory Burst
Survival Rate
Grant Support
ID/Acronym/Agency:
K08 NS048118-01A1/NS/NINDS NIH HHS; K08 NS048118-02/NS/NINDS NIH HHS; K08 NS048118-03/NS/NINDS NIH HHS; K08 NS048118-04/NS/NINDS NIH HHS; K08 NS048118-05/NS/NINDS NIH HHS; K08 NS048118-06/NS/NINDS NIH HHS; K08 NS48118/NS/NINDS NIH HHS; R01 CA095274/CA/NCI NIH HHS; R01 CA095274-09/CA/NCI NIH HHS; R01 NS057221-01A1/NS/NINDS NIH HHS; R01 NS057221-02/NS/NINDS NIH HHS; R01 NS057221-03/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Oncogene Proteins, Fusion; 0/promyelocytic leukemia-retinoic acid receptor alpha fusion oncoprotein; EC 2.1.1.37/DNA (Cytosine-5-)-Methyltransferase; EC 2.1.1.37/DNA methyltransferase 3A
Comments/Corrections
Erratum In:
Cancer Res. 2011 Apr 1;71(7):2805

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Brick1 is an essential regulator of actin cytoskeleton required for embryonic development and cell t...
Next Document:  Tumor-reactive CD8+ early effector T cells identified at tumor site in primary and metastatic melano...