Document Detail


PGE(2) transiently enhances DC expression of CCR7 but inhibits the ability of DCs to produce CCL19 and attract naive T cells.
MedLine Citation:
PMID:  20498301     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Prostaglandin E(2) (PGE(2)) is an inflammatory mediator often used to increase CCR7 expression in the dendritic cells (DCs) used as cancer vaccines and to enhance their responsiveness to lymph node-associated chemokines. Here, we show that high surface expression of CCR7 on PGE(2)-matured DCs is associated with their suppressed production of the endogenous CCR7 ligand, CCL19, and is reversible by exogenous CCL19. In contrast to the PGE(2)-matured DCs, DCs matured in the presence of toll-like receptor (TLR) ligands and interferons produce high levels of both CCL19 and CCR7 mRNA/protein, but show selectively reduced expression of surface CCR7, which is compensated after DC removal from the CCL19-rich maturation environment. In accordance with these findings, PGE(2)-matured DCs show significantly higher in vitro migratory responsiveness to lymph node-associated chemokines directly after DC generation, but not after additional short-term culture in vitro, nor in vivo in patients injected with (111)indium-labeled DCs. The differences in CCL19-producing ability imprinted during DC maturation result in their different abilities to attract CCR7(+) naive T cells. Our data help to explain the impact of PGE(2) on CCR7 expression in maturing DCs and demonstrate a novel mechanism of regulatory activity of PGE(2), mediated by the inhibition of DCs ability to attract naive T cells.
Authors:
Ravikumar Muthuswamy; Jan Mueller-Berghaus; Uwe Haberkorn; Todd A Reinhart; Dirk Schadendorf; Pawel Kalinski
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-05-24
Journal Detail:
Title:  Blood     Volume:  116     ISSN:  1528-0020     ISO Abbreviation:  Blood     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-09-03     Completed Date:  2010-10-07     Revised Date:  2011-09-13    
Medline Journal Info:
Nlm Unique ID:  7603509     Medline TA:  Blood     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1454-9     Citation Subset:  AIM; IM    
Affiliation:
Department of Surgery, University of Pittsburgh, PA 15213-1863, USA.
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MeSH Terms
Descriptor/Qualifier:
Blotting, Western
Cell Adhesion
Cell Movement
Chemokine CCL19 / metabolism*
Dendritic Cells / drug effects*,  metabolism
Dinoprostone / pharmacology*
Enzyme-Linked Immunosorbent Assay
Humans
Leukocytes, Mononuclear / cytology,  drug effects,  metabolism
Lymph Nodes / cytology,  drug effects,  metabolism
Melanoma / drug therapy,  immunology,  metabolism
Monocytes / cytology,  drug effects,  metabolism
Receptors, CCR7 / metabolism*
T-Lymphocytes / physiology*
Toll-Like Receptors
Grant Support
ID/Acronym/Agency:
CA121973/CA/NCI NIH HHS; CA132714/CA/NCI NIH HHS; CA134633/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/CCR7 protein, human; 0/Chemokine CCL19; 0/Receptors, CCR7; 0/Toll-Like Receptors; 363-24-6/Dinoprostone
Comments/Corrections

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