Document Detail


PERK in beta cell biology and insulin biogenesis.
MedLine Citation:
PMID:  20850340     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
PERK (EIF2AK3) was originally discovered as a major component of the unfolded protein response (UPR). PERK deficiency results in permanent neonatal diabetes, which was initially thought to be caused by a failure to regulate ER stress in insulin-secreting beta cells, culminating in beta cell death. However, subsequent studies found that low beta cell mass was a result of reduced cell proliferation, rather than increased apoptosis. Genetic and cellular studies of Perk-deficient beta cells showed that PERK was crucially required for ER functions including proinsulin trafficking and quality control, unrelated to the ER stress pathway. Under normal physiological conditions, changes in ER calcium levels, mediated by glucose and other insulin secretagogues, regulate PERK activity for the purpose of controlling insulin biogenesis.
Authors:
Douglas R Cavener; Sounak Gupta; Barbara C McGrath
Publication Detail:
Type:  Journal Article; Review     Date:  2010-09-17
Journal Detail:
Title:  Trends in endocrinology and metabolism: TEM     Volume:  21     ISSN:  1879-3061     ISO Abbreviation:  Trends Endocrinol. Metab.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-11-24     Completed Date:  2011-03-15     Revised Date:  2011-12-21    
Medline Journal Info:
Nlm Unique ID:  9001516     Medline TA:  Trends Endocrinol Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  714-21     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Department of Biology, Center for Cellular Dynamics, Pennsylvania State University, University Park, PA 16802, USA. drc9@psu.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Humans
Insulin / biosynthesis*
Insulin-Secreting Cells / enzymology*
Models, Biological
eIF-2 Kinase / genetics,  metabolism*
Grant Support
ID/Acronym/Agency:
R01 DK062049-03/DK/NIDDK NIH HHS; R01 DK088140-02/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Insulin; EC 2.7.10.-/PERK kinase; EC 2.7.11.1/eIF-2 Kinase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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