Document Detail


Oxygen metabolites from lavage and interstitial lung cells after inhalation of endotoxin in guinea pigs.
MedLine Citation:
PMID:  7950404     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Airborne endotoxins play a role in a variety of occupational diseases such as byssinosis and humidifier's disease and are associated with pulmonary and systemic symptoms. An excess generation of oxygen free radicals (including superoxide anion, O2-) by inflammatory cells has been suggested in endotoxemia. We have studied the release of superoxide from guinea-pig lung lavage and interstitial cells and blood monocytes (BMs) at different times after an acute inhalation exposure to bacterial endotoxin. O2- generation was measured by the O2- dismutase-inhibitable reduction of ferricytochrome c, after stimulation with phorbol-myristate-acetate (PMA) or opsonized zymosan (OZ). After endotoxin exposure, the spontaneous release of O2- remained unchanged for the three cell types. From 4 h after exposure until 48 h afterwards, lung lavage cells produced more O2- after PMA or OZ stimulation than did cells from unexposed guinea pigs. The pattern of O2- generation by interstitial cells followed that of lung lavage macrophages. O2- production remained unchanged in BMs at all times. These results suggest that endotoxin inhalation induced a priming of lavage and interstitial cells, mainly macrophages, associated with an increase in O2- production. As O2- production by BMs remained unchanged, this cell is unlikely to be responsible for the systemic symptoms seen after endotoxin inhalation.
Authors:
V Hsieh; B Amoruso-Marchat; R Rylander; B S Polla
Related Documents :
19718054 - Reactivating hif prolyl hydroxylases under hypoxia results in metabolic catastrophe and...
3165394 - Peripheral pmn cells in juvenile periodontitis. increased release of elastase and of ox...
20228054 - Protection of cells in physiological oxygen tensions against dna damage-induced apoptosis.
9576744 - Protection from reoxygenation injury by inhibition of rac1.
3913634 - Systemic lymphadenopathy (las) in intravenous drug abusers. histology, immunohistochemi...
14523124 - Investigation of the role of a beta(1-4) agarase produced by pseudoalteromonas gracilis...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  International archives of allergy and immunology     Volume:  104     ISSN:  1018-2438     ISO Abbreviation:  Int. Arch. Allergy Immunol.     Publication Date:  1994 May 
Date Detail:
Created Date:  1994-11-28     Completed Date:  1994-11-28     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  9211652     Medline TA:  Int Arch Allergy Immunol     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  42-7     Citation Subset:  IM    
Affiliation:
Institute of Social and Preventive Medicine, University Hospital, Geneva, Switzerland.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Administration, Inhalation
Animals
Bronchoalveolar Lavage Fluid / cytology
Cell Count / drug effects
Endotoxins / toxicity*
Guinea Pigs
Lung / cytology*,  drug effects*,  metabolism
Macrophages, Alveolar / drug effects,  metabolism
Monocytes / drug effects
Reactive Oxygen Species / metabolism*
Superoxides / metabolism
Tetradecanoylphorbol Acetate / pharmacology
Chemical
Reg. No./Substance:
0/Endotoxins; 0/Reactive Oxygen Species; 11062-77-4/Superoxides; 16561-29-8/Tetradecanoylphorbol Acetate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Induction of inflammatory mediator release (12-hydroxyeicosatetraenoic acid) from human platelets by...
Next Document:  Immunophenotype of lymphocytes in pericardial fluid from patients with different forms of heart dise...