Document Detail

Oxygen deficit is not affected by the rate of transition from rest to submaximal exercise.
MedLine Citation:
PMID:  2735198     Owner:  NLM     Status:  MEDLINE    
Five subjects cycled on an ergometer at power outputs corresponding to 20, 40, 60 and 80% of their maximal oxygen uptake (VO2 max). On one occasion the transition from rest to work was direct (D), while on the other occasion the power output was increased slowly (S) in a stepwise manner for 6-15 min prior to exercise at the predetermined intensity. Oxygen uptake (VO2) was measured, and O2 deficit and O2 debt were calculated. Oxygen deficit increased with the exercise intensities, the peak values being 2.1 +/- 0.2 and 1.9 +/- 0.1 litres (mean +/- SEM) at 80% of VO2 max after D and S respectively. No significant difference was observed in O2 deficit or O2 debt between D and S at any exercise intensity (P less than 0.05). The O2 debt was similar to the O2 deficit at 20, 40 and 60% of VO2 max but lower than the O2 deficit (P less than 0.05) at 80% of VO2 max. Femoral venous blood lactate remained unchanged at 20% of VO2 max but increased at the higher exercise intensities, reaching peak values of 7.6 +/- 0.6 and 7.4 +/- 1.1 mmol l-1 at 80% of VO2 max after D and S respectively. Blood lactate was not significantly different between D and S at any exercise intensity (P greater than 0.05). It is concluded that O2 deficit, O2 debt and blood lactate are not affected by the rate of transition from rest to submaximal exercise. The data contradict the hypothesis that O2 deficit is caused by an inadequate O2 transport at the onset of exercise.
J M Ren; S Broberg; K Sahlin
Related Documents :
10903158 - Patterns of cardiovascular and ventilatory response to elevated metabolic states in the...
8125878 - Effects of hyperoxia on maximal leg o2 supply and utilization in men.
3710978 - Pulmonary gas exchange in humans during exercise at sea level.
14572008 - Chamber for indirect calorimetry with accurate measurement and time discrimination of m...
23598418 - Exercise therapy augments the ischemia-induced proangiogenic state and results in susta...
21401298 - The b2 receptor of bradykinin is not essential for the post-exercise increase in glucos...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Acta physiologica Scandinavica     Volume:  135     ISSN:  0001-6772     ISO Abbreviation:  Acta Physiol. Scand.     Publication Date:  1989 Apr 
Date Detail:
Created Date:  1989-07-20     Completed Date:  1989-07-20     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  0370362     Medline TA:  Acta Physiol Scand     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  545-8     Citation Subset:  IM; S    
Department of Clinical Physiology, Huddinge University Hospital, Sweden.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Heart Rate
Lactates / blood
Oxygen Consumption*
Physical Exertion*
Reg. No./Substance:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

Previous Document:  Effects of growth hormone on skeletal muscle. I. Studies on normal adult rats.
Next Document:  Decreased level of cardiac antioxidants in endurance-trained rats.