Document Detail


Overview: hypertension and atherosclerosis.
MedLine Citation:
PMID:  3293398     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Many large-scale trials of drug treatment for hypertension have shown an apparent lack of benefit with regard to the incidence of complications related to atherosclerosis. Recent experimental evidence has contributed to an understanding of the pathogenesis of hypertensive vascular disease, although exact mechanisms by which hypertension and antihypertensive drugs influence the atherosclerotic process are still poorly defined. In animal models, hypertension appears to induce a sequence of changes in endothelial cells, smooth muscle cells, and endothelial permeability. However, increased lipid deposition in the intima and acceleration of atherosclerosis appear to require elevation of plasma lipid levels. Some beta-blockers have been found to retard the development of atherosclerosis in the cholesterol-fed rabbit model. The clinical applicability of these interesting research findings awaits further investigation.
Authors:
A Chobanian
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  American heart journal     Volume:  116     ISSN:  0002-8703     ISO Abbreviation:  Am. Heart J.     Publication Date:  1988 Jul 
Date Detail:
Created Date:  1988-08-12     Completed Date:  1988-08-12     Revised Date:  2006-02-27    
Medline Journal Info:
Nlm Unique ID:  0370465     Medline TA:  Am Heart J     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  319-22     Citation Subset:  AIM; IM    
Affiliation:
Cardiovascular Institute, Boston University School of Medicine, MA 02118.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antihypertensive Agents / therapeutic use
Arteriosclerosis / blood,  etiology*,  prevention & control
Blood Pressure / drug effects
Disease Models, Animal
Humans
Hypertension / blood,  complications*,  drug therapy
Lipids / blood
Lipoproteins / blood
Chemical
Reg. No./Substance:
0/Antihypertensive Agents; 0/Lipids; 0/Lipoproteins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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