Document Detail


Overexpression of cytochrome P450 NADPH reductase sensitises MDA 231 breast carcinoma cells to 5-fluorouracil: possible mechanisms involved.
MedLine Citation:
PMID:  18191533     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Activity of cytochromes P450 is highly dependent on cytochrome P450 NADPH reductase (P450R), but this enzyme can also metabolise drugs on its own. MDA 231 breast adenocarcinoma cells transfected with human P450R (MDA R4) or an empty vector (MDA EV) were exposed to a series of commonly used chemotherapeutic drugs. Overexpression of P450R did not affect cell sensitivity to cisplatin, mitoxantrone, paclitaxel, docetaxel, vincristine or etoposide. However, MDA R4 cells showed increased sensitivity to mitomycin C (6.6-fold) and also to 5-fluorouracil (2.8-fold). In vitro toxicity assays where mitomycin C, 5-fluorouracil and vincristine were preincubated with microsomes expressing recombinant P450R showed that this effect was not a result of direct metabolism by P450R. Levels of NADPH were considerably decreased in MDA R4 as compared to MDA EV cells, while reactive oxygen species (ROS) production was increased in MDA R4 cells in basal conditions, showing no significant further increase after treatment with mitomycin C or 5-fluorouracil. P450R overexpression appears therefore to be detrimental to MDA 231 cells, depleting NADPH and increasing ROS levels; the increased oxidative stress observed in MDA R4 cells might explain the enhanced sensitivity to 5-fluorouracil. Expression of this enzyme in tumour cells might therefore modulate response to 5-fluorouracil.
Authors:
Vanesa G Martinez; Kaye J Williams; Ian J Stratford; Martin Clynes; Robert O'Connor
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-12-07
Journal Detail:
Title:  Toxicology in vitro : an international journal published in association with BIBRA     Volume:  22     ISSN:  0887-2333     ISO Abbreviation:  Toxicol In Vitro     Publication Date:  2008 Apr 
Date Detail:
Created Date:  2008-03-10     Completed Date:  2008-07-10     Revised Date:  2009-04-10    
Medline Journal Info:
Nlm Unique ID:  8712158     Medline TA:  Toxicol In Vitro     Country:  England    
Other Details:
Languages:  eng     Pagination:  582-8     Citation Subset:  IM    
Affiliation:
National Institute for Cellular Biotechnology (NICB), Dublin City University, Glasnevin, Dublin 9, Ireland. vanesa.martinez2@mail.dcu.ie
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MeSH Terms
Descriptor/Qualifier:
Antibiotics, Antineoplastic / metabolism,  pharmacology
Antimetabolites, Antineoplastic / metabolism,  pharmacology*
Antineoplastic Agents, Phytogenic / metabolism,  pharmacology
Blotting, Western
Breast Neoplasms / drug therapy*,  genetics
Cell Line, Tumor
Cell Survival
Female
Fluorouracil / metabolism,  pharmacology*
Glutathione / metabolism
Humans
Microsomes / metabolism
Mitomycin / metabolism,  pharmacology
NADP / metabolism
NADPH-Ferrihemoprotein Reductase / biosynthesis*,  genetics,  physiology
Reactive Oxygen Species / metabolism
Transfection
Vincristine / metabolism,  pharmacology
Grant Support
ID/Acronym/Agency:
//Medical Research Council
Chemical
Reg. No./Substance:
0/Antibiotics, Antineoplastic; 0/Antimetabolites, Antineoplastic; 0/Antineoplastic Agents, Phytogenic; 0/Reactive Oxygen Species; 50-07-7/Mitomycin; 51-21-8/Fluorouracil; 53-59-8/NADP; 57-22-7/Vincristine; 70-18-8/Glutathione; EC 1.6.2.4/NADPH-Ferrihemoprotein Reductase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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