Document Detail


Over-expression of hTERT in CHO K1 results in decreased apoptosis and reduced serum dependency.
MedLine Citation:
PMID:  16144725     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The enzyme telomerase plays a crucial role in cellular proliferation. By adding hexameric repeats to the chromosome ends, it prevents telomeric loss and, thus entry into senescence of limited life span cells. It is unclear, however, what would be the effect of over-expressing telomerase in an immortalised cell line, characterised by unlimited life span and high levels of apoptosis under sub-optimal growth conditions. In order to address this question, we have transfected the immortal cell line CHO K1 with the human telomerase reverse transcriptase (hTERT) catalytic subunit. Differences in the growth profile and apoptosis levels between the cells over-expressing hTERT (Telo) and the cells containing mock vector were found under standard growth conditions. Similarly, the Telo cells showed lower levels of apoptosis, greater attachment tendency and higher viable cell density under serum-deprived conditions compared to the control cell line, suggesting a major role for hTERT over-expression in stressed cultures. Using a mouse cDNA microarray, the collagen type III and V genes were shown to have at least a 10-fold higher expression in the Telo cells than the control cells, suggesting a role of hTERT in the cell attachment pathways.
Authors:
Francesco Crea; Donatella Sarti; Francesco Falciani; Mohamed Al-Rubeai
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Publication Detail:
Type:  Journal Article     Date:  2005-09-06
Journal Detail:
Title:  Journal of biotechnology     Volume:  121     ISSN:  0168-1656     ISO Abbreviation:  J. Biotechnol.     Publication Date:  2006 Jan 
Date Detail:
Created Date:  2006-01-09     Completed Date:  2006-06-21     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8411927     Medline TA:  J Biotechnol     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  109-23     Citation Subset:  IM    
Affiliation:
Department of Chemical Engineering, School of Engineering, University of Birmingham, Birmingham B15 2TT, UK.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / genetics*
CHO Cells
Catalytic Domain / genetics
Cell Adhesion / genetics
Cell Transformation, Neoplastic / genetics*
Collagen Type III / biosynthesis
Collagen Type V / biosynthesis
Cricetinae
Cricetulus
DNA-Binding Proteins / biosynthesis*,  genetics
Gene Expression Regulation, Neoplastic / genetics
Humans
Recombinant Proteins / biosynthesis*,  genetics
Telomerase / biosynthesis*,  genetics
Chemical
Reg. No./Substance:
0/Collagen Type III; 0/Collagen Type V; 0/DNA-Binding Proteins; 0/Recombinant Proteins; EC 2.7.7.49/Telomerase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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