Document Detail


Osthol regulates hepatic PPAR alpha-mediated lipogenic gene expression in alcoholic fatty liver murine.
MedLine Citation:
PMID:  20042322     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Our previous studies found that osthol, an active constituent isolated from Cnidium monnieri (L.) Cusson (Apiaceae), could ameliorate the accumulation of lipids and decrease the lipid levels in serum and hepatic tissue in alcohol-induced fatty liver mice and rats. The objective of this study was to investigate its possible mechanism of the lipid-lowering effect. A mouse model with alcoholic fatty liver was induced by orally feeding 52% erguotou wine by gavage when they were simultaneously treated with osthol 10, 20, 40 mg/kg for 4 weeks. The BRL cells (rat hepatocyte line) were cultured and treated with osthol at 25, 50, 100, 200 microg/ml for 24h. The mRNA expressions of peroxisome proliferator-activated receptor (PPAR) alpha, diacylglycerol acyltransferase (DGAT), 3-hydroxy-3-methylglutaryl-CoA (HMG-CoA) reductase and cholesterol 7 alpha-hydroxylase (CYP7A) in mouse hepatic tissue or cultured hepatocytes were determined by reverse transcription polymerase chain reaction (RT-PCR). After treatment with osthol, the PPAR alpha mRNA expression in mouse liver and cultured hepatocytes was increased in dose dependent manner, while its related target genes for mRNA expression, e.g., DGAT and HMG-CoA reductase, were decreased, the CYP7A was inversely increased. And osthol-regulated mRNA expressions of DGAT, HMG-CoA reductase and CYP7A in the cultured hepatocytes were abrogated after pretreatment with specific inhibitor of PPAR alpha, MK886. It was concluded that osthol might regulate the gene expressions of DGAT, HMG-CoA reductase and CYP7A via increasing the PPAR alpha mRNA expression.
Authors:
Fan Sun; Mei-lin Xie; Jie Xue; Heng-bin Wang
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-12-29
Journal Detail:
Title:  Phytomedicine : international journal of phytotherapy and phytopharmacology     Volume:  17     ISSN:  1618-095X     ISO Abbreviation:  Phytomedicine     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-05-24     Completed Date:  2010-12-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9438794     Medline TA:  Phytomedicine     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  669-73     Citation Subset:  IM    
Copyright Information:
(c) 2009 Elsevier GmbH. All rights reserved.
Affiliation:
Department of Pharmacology, Medical College of Soochow University, Suzhou 215123, Jiangsu Province, China.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cholesterol 7-alpha-Hydroxylase / genetics,  metabolism
Cnidium / chemistry*
Coumarins / pharmacology*
Diacylglycerol O-Acyltransferase / genetics,  metabolism
Disease Models, Animal
Dose-Response Relationship, Drug
Fatty Liver, Alcoholic / drug therapy,  genetics,  metabolism*
Fruit
Gene Expression / drug effects*
Hepatocytes / drug effects,  metabolism
Hydroxymethylglutaryl CoA Reductases / genetics,  metabolism
Hypolipidemic Agents / pharmacology*
Liver / drug effects*,  metabolism,  pathology
Male
Mice
Mice, Inbred Strains
PPAR alpha / genetics,  metabolism*
Phytotherapy
RNA, Messenger / metabolism
Reverse Transcriptase Polymerase Chain Reaction
Chemical
Reg. No./Substance:
0/Coumarins; 0/Hypolipidemic Agents; 0/PPAR alpha; 0/RNA, Messenger; 484-12-8/osthol; EC 1.1.1.-/Hydroxymethylglutaryl CoA Reductases; EC 1.14.13.17/Cholesterol 7-alpha-Hydroxylase; EC 2.3.1.20/Diacylglycerol O-Acyltransferase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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