Document Detail


Osteoblasts respond to pulsatile fluid flow with short-term increases in PGE(2) but no change in mineralization.
MedLine Citation:
PMID:  11299276     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Although there is no consensus as to the precise nature of the mechanostimulatory signals imparted to the bone cells during remodeling, it has been postulated that deformation-induced fluid flow plays a role in the mechanotransduction pathway. In vitro, osteoblasts respond to fluid shear stress with an increase in PGE(2) production; however, the long-term effects of fluid shear stress on cell proliferation and differentiation have not been examined. The goal of this study was to apply continuous pulsatile fluid shear stresses to osteoblasts and determine whether the initial production of PGE(2) is associated with long-term biochemical changes. The acute response of bone cells to a pulsatile fluid shear stress (0.6 +/- 0.5 Pa, 3.0 Hz) was characterized by a transient fourfold increase in PGE(2) production. After 7 days of static culture (0 dyn/cm(2)) or low (0.06 +/- 0.05 Pa, 0.3 Hz) or high (0.6 +/- 0.5 Pa, 3.0 Hz) levels of pulsatile fluid shear stress, the bone cells responded with an 83% average increase in cell number, but no statistical difference (P > 0.53) between the groups was observed. Alkaline phosphatase activity per cell decreased in the static cultures but not in the low- or high-flow groups. Mineralization was also unaffected by the different levels of applied shear stress. Our results indicate that short-term changes in PGE(2) levels caused by pulsatile fluid flow are not associated with long-term changes in proliferation or mineralization of bone cells.
Authors:
E A Nauman; R L Satcher; T M Keaveny; B P Halloran; D D Bikle
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.    
Journal Detail:
Title:  Journal of applied physiology (Bethesda, Md. : 1985)     Volume:  90     ISSN:  8750-7587     ISO Abbreviation:  J. Appl. Physiol.     Publication Date:  2001 May 
Date Detail:
Created Date:  2001-04-12     Completed Date:  2001-07-05     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8502536     Medline TA:  J Appl Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1849-54     Citation Subset:  IM; S    
Affiliation:
Orthopaedic Biomechanics Laboratory, Department of Mechanical Engineering, University of California, Berkeley 94720-1740, USA. enauman@tulane.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Calcification, Physiologic / physiology*
Cell Differentiation
Cell Division
Cells, Cultured
Dinoprostone / biosynthesis*
Femur
Kinetics
Male
Osteoblasts / cytology*,  physiology*
Rats
Rats, Sprague-Dawley
Stress, Mechanical
Tibia
Time Factors
Chemical
Reg. No./Substance:
363-24-6/Dinoprostone
Investigator
Investigator/Affiliation:
B P Halloran / U CA, San Francisco; D D Bikle / U CA, San Francisco

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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