Document Detail


Organelle dynamics and membrane trafficking in apoptosis and autophagy.
MedLine Citation:
PMID:  20865668     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The accurate control of cell death is a vital aspect of development in metazoans and plays crucial roles in the prevention of disease. Apoptosis is the main form of regulated cell death in multicellular organisms, although there are other contributory pathways. During apoptosis, mammalian cells undergo dramatic changes in organelle structure ad organisation that define the apoptotic execution phase. Although the roles of apoptotic protease machinery (the caspases) in these rearrangements are quite well understood, the purpose of organelle disruption during cell death is not yet entirely appreciated. Indeed, recent evidence implicates caspase targeting of organellar proteins and subsequent organelle disruption upstream of apoptotic execution proper, suggesting the existence of pathways linking organelle damage to cell death. In this review, we describe the changes to the endomembrane system that are inherent during the apoptotic execution phase, and examine the evidence for endomembrane-mediated pathways towards apoptotic execution. We also discuss aspects of the molecular control of autophagy - an important contributor to a cell's response to stress, and a membrane trafficking process whose regulation is linked to the apoptotic machinery at multiple levels.
Authors:
Jade P X Cheng; Jon D Lane
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Histology and histopathology     Volume:  25     ISSN:  1699-5848     ISO Abbreviation:  Histol. Histopathol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-09-24     Completed Date:  2011-01-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8609357     Medline TA:  Histol Histopathol     Country:  Spain    
Other Details:
Languages:  eng     Pagination:  1457-72     Citation Subset:  IM    
Affiliation:
Cell Biology Laboratories, Department of Biochemistry, University of Bristol, School of Medical and Veterinary Sciences, University Walk, Bristol, UK.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / physiology*
Autophagy / physiology*
Cell Membrane / physiology*
Humans
Organelles / physiology*
Protein Transport / physiology*
Signal Transduction / physiology*
Grant Support
ID/Acronym/Agency:
//Biotechnology and Biological Sciences Research Council

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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