Document Detail


Optimal pressure for low pressure controlled reperfusion to efficiently protect ischemic heart: an experimental study in rats.
MedLine Citation:
PMID:  19328961     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recent work has demonstrated the benefit of low pressure (LP) reperfusion to protect the heart undergoing an ischemic insult. The goal of the present study was to determine the optimal pressure for the application of LP reperfusion. Isolated rats hearts (n = 30) were exposed to 40 minutes of global warm ischemia followed by 70 minutes of reperfusion with a pressure fixed at 100 cm H(2)O (normal pressure [NP] = control group), 85 cm (group LP [low pressure]-85), 70 cm (group LP-70), or 55 cm (group LP-55). Cardiac function was assessed during reperfusion using the Langendorff model. Myocardial necrosis was assessed by measuring lactate dehydrogenase (LDH) and creatine kinase (CK) leakage in the coronary effluents. Functional recovery was progressively and significantly improved with decreased perfusion pressure. Rate-pressure product (RPP) averaged 3765 +/- 408, 6824 +/- 439, and 12,036 +/- 664 mm Hg/min, respectively, among the control, LP-85, and LP-70 groups (P < .001, LP-70 vs other groups). However, RPP collapsed in the LP-55 group. Similarly, necrosis as measured by LDH and CK leakage progressively reduced between LP-100 and LP-70 hearts (P < .01), with a drastic increase in enzyme in the LP-55 group. In conclusion, this study demonstrated that 70 cm H(2)O is an optimal LP to improve postischemic contractile dysfunction and attenuate necrosis during reperfusion.
Authors:
C Nemlin; S Benhabbouche; J C Bopassa; L Sebbag; M Ovize; R Ferrera
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Transplantation proceedings     Volume:  41     ISSN:  0041-1345     ISO Abbreviation:  Transplant. Proc.     Publication Date:  2009 Mar 
Date Detail:
Created Date:  2009-03-30     Completed Date:  2009-07-29     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0243532     Medline TA:  Transplant Proc     Country:  United States    
Other Details:
Languages:  eng     Pagination:  703-4     Citation Subset:  IM    
Affiliation:
INSERM U886 and Université Claude Bernard Lyon-1, Lyon, France.
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MeSH Terms
Descriptor/Qualifier:
Animals
Diastole
Disease Models, Animal
L-Lactate Dehydrogenase / blood
Male
Myocardial Ischemia / pathology,  prevention & control*
Myocardial Reperfusion / methods*
Pressure
Rats
Rats, Wistar
Reperfusion Injury / pathology
Ventricular Function, Left / physiology
Chemical
Reg. No./Substance:
EC 1.1.1.27/L-Lactate Dehydrogenase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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