Document Detail


Opposing effects of dietary sugar and saturated fat on cardiovascular risk factors and glucose metabolism in mitochondrially impaired mice.
MedLine Citation:
PMID:  20221766     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
PURPOSE: Both dietary fat and dietary sucrose are major components of Western diets that may differentially affect the risk for body mass gain, diabetes mellitus, and cardiovascular disease.
METHODS: We have phenotypically analyzed mice with ubiquitously impaired expression of mitochondrial frataxin protein that were challenged with diets differing in macronutrient content, namely high-sucrose/low-fat and high-saturated fat/low-sugar diets.
RESULTS: We find here that a high-sucrose/low-fat diet has especially detrimental effects in mice with impaired mitochondrial metabolism promoting several independent cardiovascular risk factors, including impaired glucose metabolism, fasting hyperinsulinemia, reduced glucose-stimulated insulin secretion, increased serum triglycerides, and elevated cholesterol levels due to increased expression of HMG-CoA reductase. In contrast, a high-saturated fat/low-sugar diet protects mice with impaired mitochondrial metabolism from diet-induced obesity by increasing total energy expenditure and increasing expression of ACAA2, a rate-limiting enzyme of mitochondrial beta-oxidation, whereas no concomitant improvement of glucose metabolism was observed.
CONCLUSIONS: Taken together, our results suggest that mitochondrial dysfunction may cause sucrose to become a multifunctional cardiovascular risk factor, whereas low-sugar diets high in saturated fat may prevent weight gain without improving glucose metabolism.
Authors:
Doreen Kuhlow; Kim Zarse; Anja Voigt; Tim J Schulz; Klaus J Petzke; Lutz Schomburg; Andreas F H Pfeiffer; Michael Ristow
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-03-10
Journal Detail:
Title:  European journal of nutrition     Volume:  49     ISSN:  1436-6215     ISO Abbreviation:  Eur J Nutr     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-09-24     Completed Date:  2011-01-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100888704     Medline TA:  Eur J Nutr     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  417-27     Citation Subset:  IM    
Affiliation:
Department of Human Nutrition, Institute of Nutrition, University of Jena, 07743 Jena, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cardiovascular Diseases / diet therapy*
Cardiovascular System / metabolism
Cholesterol / blood,  metabolism
Diabetes Mellitus / diet therapy
Diet, Fat-Restricted
Dietary Fats / blood*,  metabolism
Dietary Sucrose / blood*,  metabolism
Gene Knockdown Techniques
Glucose / metabolism*
Insulin / secretion
Iron-Binding Proteins / metabolism
Male
Mice
Mitochondria / metabolism*
Obesity / diet therapy
Oxidation-Reduction
Risk Factors
Triglycerides / blood,  metabolism
Weight Gain
Chemical
Reg. No./Substance:
0/Dietary Fats; 0/Dietary Sucrose; 0/Iron-Binding Proteins; 0/Triglycerides; 0/frataxin; 11061-68-0/Insulin; 50-99-7/Glucose; 57-88-5/Cholesterol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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