Document Detail


Nutrition and immunity: the immunoregulatory effect of n-6 essential fatty acids is mediated through prostaglandin E.
MedLine Citation:
PMID:  4040494     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Suppression of cell-mediated immune responses by essential fatty acids (EFA) was demonstrated in mice maintained on a standard laboratory diet for rodents. Daily oral administration of EFA at doses ranging from 125 to 750 mg/kg body weight significantly suppressed local host-versus-graft and graft-versus-host reactions as measured by popliteal lymph node assay. Studies employing immune sera directed against E-type prostaglandin demonstrated that n-6 EFA-induced suppression was mediated through prostaglandin E1. In titration experiments the effect of n-6 EFA on the reactions was dose dependent with enhancement of the responses at low concentrations and suppression at high concentrations.
Authors:
J Mertin; A Stackpoole; S Shumway
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  International archives of allergy and applied immunology     Volume:  77     ISSN:  0020-5915     ISO Abbreviation:  Int. Arch. Allergy Appl. Immunol.     Publication Date:  1985  
Date Detail:
Created Date:  1985-09-16     Completed Date:  1985-09-16     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  0404561     Medline TA:  Int Arch Allergy Appl Immunol     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  390-5     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Alprostadil
Animals
Antibodies / immunology
Fatty Acids, Essential / immunology*,  pharmacology
Graft vs Host Reaction / drug effects
Host vs Graft Reaction / drug effects
Immunity, Cellular* / drug effects
Immunosuppression
Mice
Mice, Inbred Strains
Nutritional Physiological Phenomena*
Prostaglandins E / immunology*
Spleen / transplantation
Transplantation, Homologous
Chemical
Reg. No./Substance:
0/Antibodies; 0/Fatty Acids, Essential; 0/Prostaglandins E; 745-65-3/Alprostadil

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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