Document Detail


Nonsteroidal selective androgen receptor modulators enhance female sexual motivation.
MedLine Citation:
PMID:  20444881     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Women experience a decline in estrogen and androgen levels after natural or surgically induced menopause, effects that are associated with a loss of sexual desire and bone mineral density. Studies in our laboratories have shown the beneficial effects of selective androgen receptor modulators (SARMs) in the treatment of osteoporosis and muscle wasting in animal models. A series of S-3-(phenoxy)-2-hydroxy-2-methyl-N-(4-cyano-3-trifluoromethyl-phenyl)-propionamide analogs was synthesized to evaluate the effects of B-ring substitutions on in vitro and in vivo pharmacologic activity, especially female sexual motivation. The androgen receptor (AR) relative binding affinities ranged from 0.1 to 26.5% (relative to dihydrotestosterone) and demonstrated a range of agonist activity at 100 nM. In vivo pharmacologic activity was first assessed by using male rats. Structural modifications to the B-ring significantly affected the selectivity of the SARMs, demonstrating that single-atom substitutions can dramatically and unexpectedly influence activity in androgenic (i.e., prostate) and anabolic (i.e., muscle) tissues. (S)-N-(4-cyano-3-trifluoromethyl-phenyl)-3-(3-fluoro,4-chlorophenoxy)-2-hydroxy-2-methyl-propanamide (S-23) displayed full agonist activity in androgenic and anabolic tissues; however, the remaining SARMs were more prostate-sparing, selectively maintaining the size of the levator ani muscle in castrated rats. The partner-preference paradigm was used to evaluate the effects of SARMs on female sexual motivation. With the exception of two four-halo substituted analogs, the SARMs increased sexual motivation in ovariectomized rats, with potency and efficacy comparable with testosterone propionate. These results indicate that the AR is important in regulating female libido given the nonaromatizable nature of SARMs and it could be a superior alternative to steroidal testosterone preparations in the treatment of hypoactive sexual desire disorder.
Authors:
Amanda Jones; Dong Jin Hwang; Charles B Duke; Yali He; Anjaiah Siddam; Duane D Miller; James T Dalton
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-05-05
Journal Detail:
Title:  The Journal of pharmacology and experimental therapeutics     Volume:  334     ISSN:  1521-0103     ISO Abbreviation:  J. Pharmacol. Exp. Ther.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-19     Completed Date:  2010-08-17     Revised Date:  2011-08-03    
Medline Journal Info:
Nlm Unique ID:  0376362     Medline TA:  J Pharmacol Exp Ther     Country:  United States    
Other Details:
Languages:  eng     Pagination:  439-48     Citation Subset:  IM    
Affiliation:
Division of Pharmaceutics, College of Pharmacy, Ohio State University, Columbus, Ohio, USA.
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MeSH Terms
Descriptor/Qualifier:
Androgens*
Anilides / chemistry,  pharmacology*
Animals
Binding, Competitive
Cell Line
Female
Follicle Stimulating Hormone / blood
Humans
Luteinizing Hormone / blood
Male
Orchiectomy
Organ Size
Ovariectomy
Prostate / anatomy & histology,  drug effects
Radioligand Assay
Rats
Rats, Sprague-Dawley
Receptors, Androgen / physiology*
Sex Factors
Sexual Behavior, Animal / drug effects*
Stereoisomerism
Structure-Activity Relationship
Transcriptional Activation
Uterus / anatomy & histology,  drug effects
Grant Support
ID/Acronym/Agency:
1 RO1 DK59800-08/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Androgens; 0/Anilides; 0/N-(4-cyano-3-trifluoromethylphenyl)-3-(3-fluoro-4-chlorophenoxy)-2-hydroxy-2-methylpropanamide; 0/Receptors, Androgen; 9002-67-9/Luteinizing Hormone; 9002-68-0/Follicle Stimulating Hormone
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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