Document Detail


No hyperalgesia following opioid withdrawal after the oripavine derivative etorphine compared to remifentanil and sufentanil.
MedLine Citation:
PMID:  20077594     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND AND OBJECTIVE: The concept of opioid-induced hyperalgesia has recently gained prominence as a contributing factor for long-term treatment failure. METHODS: To evaluate possible differences of opioids used in anaesthesia, cumulative doses of sufentanil and remifentanil were compared with escalating doses of the oripavine derivative etorphine, in awake and trained canines. This was followed by naloxone unmasking a possible hyperalgesic state, which had developed during opioid administration. Heart rate, blood pressure and propagation of nociceptive volleys in somatosensory-evoked potentials as well as the skin-twitch reflex were evaluated. RESULTS: Opioid-related hypotension and bradycardia were reversed by naloxone with a late (30 min) overshoot of R43 and R17% after remifentanil and sufentanil, respectively. Following etorphine, overshoot in mean blood pressure was R9%, whereas heart rate still remained below S9% when compared with control. Peak hyperalgesia, as detected in the somatosensory-evoked potential and skin-twitch, increased by R70% after remifentanil and by R43% after sufentanil. This reflected a significant (P<0.005) increase in propagation of nociceptive afferents as late as 30 min after naloxone reversal. Such potentiation was not observed in the etorphine group, as peak somatosensory-evoked potential deflection and skin-twitch remained below S80% when compared with control. CONCLUSION: The pure mu-agonists sufentanil or remifentanil seem to induce a 'bimodal' inhibitory followed by an excitatory effect. The latter is unmasked by naloxone in the postadministration period. In contrast, this is not seen with etorphine, a close congener of buprenorphine. The proposed mode of action of such hyperexcitatory effects may involve second-messenger-mediated G-protein activation, originally proposed by others. Ligands of the oripavine series may present an alternative for prevention of opioid-induced hyperalgesia in patients.
Authors:
Enno Freye; Joseph V Levy
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Publication Detail:
Type:  Comparative Study; Journal Article    
Journal Detail:
Title:  European journal of anaesthesiology     Volume:  27     ISSN:  1365-2346     ISO Abbreviation:  Eur J Anaesthesiol     Publication Date:  2010 Feb 
Date Detail:
Created Date:  2010-01-14     Completed Date:  2010-03-30     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8411711     Medline TA:  Eur J Anaesthesiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  174-80     Citation Subset:  IM    
Affiliation:
Clinics of Vascular Surgery, University Clinics of Duesseldorf, Duesseldorf, Germany. enno.freye@uni-duesseldorf.de
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MeSH Terms
Descriptor/Qualifier:
Afferent Pathways / drug effects
Analgesics, Opioid / administration & dosage,  toxicity*
Animals
Blood Pressure / drug effects
Bradycardia / chemically induced
Dogs
Dose-Response Relationship, Drug
Etorphine / administration & dosage,  toxicity*
Evoked Potentials, Somatosensory / drug effects
Heart Rate / drug effects
Hyperalgesia / chemically induced
Hypotension / chemically induced
Naloxone / pharmacology
Narcotic Antagonists / pharmacology
Piperidines / administration & dosage,  toxicity*
Substance Withdrawal Syndrome
Sufentanil / administration & dosage,  toxicity*
Chemical
Reg. No./Substance:
0/Analgesics, Opioid; 0/Narcotic Antagonists; 0/Piperidines; 132875-61-7/remifentanil; 14521-96-1/Etorphine; 465-65-6/Naloxone; 56030-54-7/Sufentanil

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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