Document Detail


Nitric oxide induces the apoptosis of human BCR-ABL-positive myeloid leukemia cells: evidence for the chelation of intracellular iron.
MedLine Citation:
PMID:  11960353     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Anti-leukemia activity of human macrophages involves the generation of nitric oxide (NO) derivatives. However, leukemic transformation may involve mechanisms that rescue cells from NO-mediated apoptosis. In the present work, we analyzed the effects of exogenous NO on the proliferation of BCR-ABL(+) chronic myelogenous leukemia (CML) cells. As normal leukocytes, the proliferation of leukemia cells was inhibited by SNAP (S-nitroso-N-acetyl-penicillamine), GEA (Oxatriazolium amino-chloride), and SIN-1 (Morpholino-sydnonimine), whereas SNP (sodium nitroprusside) had no effect on leukemia cell growth. SIN-1 induced higher anti-proliferation activity in BCR-ABL(+) cells, compared to normal hemopoietic cells. Inhibition of leukemia cell proliferation correlated with increased apoptosis and DEVDase activity. The simultaneous addition of exogenous iron reversed NO-mediated inhibition of cell growth, caspase activation and apoptosis in all BCR-ABL(+) cells tested. The quantification of intracellular iron levels in leukemia cells indicated that NO induced an early, dose-dependent decrease in ferric iron levels. Accordingly, elevation of intracellular iron protected leukemia cells from NO-mediated apoptosis. Together, the present work reveals the presence of an iron-dependant mechanism for leukemia cell rescue from NO-induced growth inhibition and apoptosis.
Authors:
H Ferry-Dumazet; M Mamani-Matsuda; M Dupouy; F Belloc; D Thiolat; G Marit; M Arock; J Reiffers; M D Mossalayi
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Leukemia     Volume:  16     ISSN:  0887-6924     ISO Abbreviation:  Leukemia     Publication Date:  2002 Apr 
Date Detail:
Created Date:  2002-04-17     Completed Date:  2002-05-07     Revised Date:  2013-03-04    
Medline Journal Info:
Nlm Unique ID:  8704895     Medline TA:  Leukemia     Country:  England    
Other Details:
Languages:  eng     Pagination:  708-15     Citation Subset:  IM    
Affiliation:
Bone Marrow Transplantation, CNRS UMR5540 and EA482, Bordeaux-2 University, Bordeaux, France.
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*,  physiology
Cell Cycle / drug effects
Cell Division / drug effects
Cells, Cultured / drug effects
Ferricyanides / pharmacology
Fusion Proteins, bcr-abl / genetics,  metabolism*
Humans
Iron / metabolism*
Leukemia, Myelogenous, Chronic, BCR-ABL Positive / metabolism*,  pathology
Nitric Oxide / pharmacology*
Nitric Oxide Donors / pharmacology
Penicillamine / analogs & derivatives,  pharmacology
S-Nitroso-N-Acetylpenicillamine / pharmacology
Signal Transduction / physiology
Chemical
Reg. No./Substance:
0/Ferricyanides; 0/Fusion Proteins, bcr-abl; 0/Nitric Oxide Donors; 10102-43-9/Nitric Oxide; 52-67-5/Penicillamine; 7439-89-6/Iron; 79032-48-7/S-Nitroso-N-Acetylpenicillamine

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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