Document Detail


Nitric oxide-endothelin-1 interaction in humans.
MedLine Citation:
PMID:  9134909     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Healthy men received NG-monomethyl-L-arginine (L-NMMA) intravenously to study cardiovascular and metabolic effects of nitric oxide synthase blockade and whether this alters the response to endothelin-1 (ET-1) infusion. Controls only received ET-1. L-NMMA effects were that heart rate (17%) cardiac output (17%), and splanchnic and renal blood flow (both 33%) fell promptly (all P < 0.01). Mean arterial blood pressure (6%), and systemic (28%) and pulmonary (40%) vascular resistance increased (P < 0.05 to 0.001). Arterial ET-1 levels (21%) increased due to a pulmonary net ET-1 release (P < 0.05 to 0.01). Splanchnic glucose output (SGO) fell (26%, P < 0.01). Arterial insulin and glucagon were unchanged. Subsequent ET-1 infusion caused no change in mean arterial pressure, heart rate, or cardiac output, as found in the present controls, or in splanchnic and renal blood flow or splanchnic glucose output as previously found with ET-1 (G. Ahlborg, E. Weitzberg, and J. M. Lundberg. J. Appl. Physiol. 79: 141-145, 1995). In conclusion, L-NMMA like ET-1, induces prolonged cardiovascular effects and suppresses SGO. L-NMMA causes pulmonary ET-1 release and blocks responses to ET-1 infusion. The results indicate that nitric oxide inhibits ET-1 production and thereby interacts with ET-1 regarding increase in vascular tone and reduction of SGO in humans.
Authors:
G Ahlborg; J M Lundberg
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of applied physiology (Bethesda, Md. : 1985)     Volume:  82     ISSN:  8750-7587     ISO Abbreviation:  J. Appl. Physiol.     Publication Date:  1997 May 
Date Detail:
Created Date:  1997-07-21     Completed Date:  1997-07-21     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8502536     Medline TA:  J Appl Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1593-600     Citation Subset:  IM    
Affiliation:
Department of Clinical Physiology, Huddinge University Hospital, Sweden.
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MeSH Terms
Descriptor/Qualifier:
Adult
Blood Gas Analysis
Blood Glucose
Blood Pressure / drug effects
Cardiac Output / drug effects
Endothelin-1 / blood*
Enzyme Inhibitors / administration & dosage
Glucagon / blood
Heart Rate / drug effects
Humans
Injections, Intravenous
Insulin / blood
Male
Muscle Contraction / physiology
Muscle, Smooth / drug effects,  enzymology
Nitric Oxide / biosynthesis,  metabolism*
Nitric Oxide Synthase / antagonists & inhibitors
Pulmonary Circulation / drug effects,  physiology
Renal Circulation / drug effects,  physiology
Respiration / physiology
Splanchnic Circulation / drug effects,  physiology
Vascular Resistance / drug effects
omega-N-Methylarginine / administration & dosage
Chemical
Reg. No./Substance:
0/Blood Glucose; 0/Endothelin-1; 0/Enzyme Inhibitors; 10102-43-9/Nitric Oxide; 11061-68-0/Insulin; 17035-90-4/omega-N-Methylarginine; 9007-92-5/Glucagon; EC 1.14.13.39/Nitric Oxide Synthase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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