Document Detail


New players in the sepsis-protective activated protein C pathway.
MedLine Citation:
PMID:  20714106     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recombinant activated protein C (aPC) improves the survival of patients with severe sepsis, but the precise molecular and cellular targets through which it mediates this effect remain incompletely understood. In this issue of the JCI, Kerschen et al. show that endothelial cell protein C receptor (EPCR) is specifically expressed by mouse CD8+ dendritic cells and that these coordinators of host responses to systemic infection are required for aPC to provide protection against the lethality of sepsis. An additional study, by Cao and colleagues, recently published in the JCI, implicates the leukocyte integrin CD11b in the pathways by which aPC mediates antiinflammatory effects in the context of lethal sepsis in mice, suggesting a common thread of synergistic control of innate immune responses by life-saving aPC therapy.
Authors:
Wolfram Ruf
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Publication Detail:
Type:  Comment; Journal Article; Research Support, N.I.H., Extramural     Date:  2010-08-16
Journal Detail:
Title:  The Journal of clinical investigation     Volume:  120     ISSN:  1558-8238     ISO Abbreviation:  J. Clin. Invest.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-09-02     Completed Date:  2010-12-15     Revised Date:  2012-02-07    
Medline Journal Info:
Nlm Unique ID:  7802877     Medline TA:  J Clin Invest     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3084-7     Citation Subset:  AIM; IM    
Affiliation:
Department of Immunology and Microbial Science, The Scripps Research Institute, La Jolla, California 92037, USA. ruf@scripps.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Antigens, CD11b / metabolism
Blood Coagulation Factors / metabolism
Forecasting
Humans
Leukocytes / metabolism
Mice
Protein C / metabolism*,  physiology*
Receptors, Cell Surface / metabolism
Recombinant Proteins / metabolism
Sepsis / immunology*,  metabolism*
Grant Support
ID/Acronym/Agency:
HL-060742/HL/NHLBI NIH HHS; HL-077753/HL/NHLBI NIH HHS; HL-31950/HL/NHLBI NIH HHS; R01 HL077753-08/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD11b; 0/Blood Coagulation Factors; 0/Protein C; 0/Receptors, Cell Surface; 0/Recombinant Proteins; 0/activated protein C receptor
Comments/Corrections
Comment On:
J Clin Invest. 2010 Sep;120(9):3167-78   [PMID:  20714108 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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