Document Detail


New developments in the regulation of intestinal copper absorption.
MedLine Citation:
PMID:  19906252     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The transition metal copper is an essential trace element involved in many enzymatic processes that require redox-chemistry. The redox-activity of copper is potentially harmful. Severe imbalance of copper homeostasis can occur with some hereditary disorders of copper metabolism. Copper is acquired from the diet by intestinal absorption and is subsequently distributed throughout the body. The regulation of intestinal copper absorption to maintain whole-body copper homeostasis is currently poorly understood. This review evaluates novel findings regarding the molecular mechanism of intestinal copper uptake. The role of recently identified transporters in enterocyte copper uptake and excretion into the portal circulation is described, and the regulation of dietary copper uptake during physiological and pathophysiological conditions is discussed.
Authors:
Peter V E van den Berghe; Leo W J Klomp
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Nutrition reviews     Volume:  67     ISSN:  1753-4887     ISO Abbreviation:  Nutr. Rev.     Publication Date:  2009 Nov 
Date Detail:
Created Date:  2009-11-12     Completed Date:  2010-01-25     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0376405     Medline TA:  Nutr Rev     Country:  United States    
Other Details:
Languages:  eng     Pagination:  658-72     Citation Subset:  IM    
Affiliation:
Department of Metabolic and Endocrine Diseases, University Medical Center Utrecht, 3584 EA Utrecht, The Netherlands.
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MeSH Terms
Descriptor/Qualifier:
Biological Transport
Ceruloplasmin / metabolism
Copper / deficiency,  metabolism,  pharmacokinetics*
Enterocytes / physiology*
Hepatolenticular Degeneration / genetics
Homeostasis
Humans
Intestinal Absorption / drug effects*
Menkes Kinky Hair Syndrome / genetics
Metallothionein / metabolism
Oxidation-Reduction
Trace Elements / pharmacokinetics*
Chemical
Reg. No./Substance:
0/Trace Elements; 7440-50-8/Copper; 9038-94-2/Metallothionein; EC 1.16.3.1/Ceruloplasmin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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