Document Detail


New developments in the discovery of small molecule Hedgehog pathway antagonists.
MedLine Citation:
PMID:  20399136     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The Hedgehog (Hh) signaling pathway is crucial for normal embryonic development. Aberrant Hh signaling is implicated in numerous pathologic conditions including proliferative diseases such as cancer. During the past decade, academic and industrial research efforts have resulted in the discovery of a variety of Hh pathway antagonists. This review focuses on the most recent advances in this field with particular emphasis on the medicinal chemistry approaches used to discover these Hh antagonists. While most of the small molecule modulators of the Hh pathway were discovered through screening and subsequent medicinal chemistry, a number of them originated from rational design or natural products.
Authors:
Martin R Tremblay; Karen McGovern; Margaret A Read; Alfredo C Castro
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Current opinion in chemical biology     Volume:  14     ISSN:  1879-0402     ISO Abbreviation:  Curr Opin Chem Biol     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-05-31     Completed Date:  2010-08-31     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9811312     Medline TA:  Curr Opin Chem Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  428-35     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Infinity Pharmaceuticals, Inc., 780 Memorial Drive, Cambridge, MA 02139, USA. martin.tremblay@infi.com
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MeSH Terms
Descriptor/Qualifier:
Drug Discovery / methods*
Drug Evaluation, Preclinical / methods
Hedgehog Proteins / antagonists & inhibitors*
Humans
Neoplasms / drug therapy
Signal Transduction
Small Molecule Libraries*
Chemical
Reg. No./Substance:
0/Hedgehog Proteins; 0/Small Molecule Libraries

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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